Systematic and Endothelial Inflammation and Endothelial Progenitor Cell Levels in Emphysematous Rats Exposed to Intermittent Hypoxia.

BACKGROUND: This study aimed to develop an overlap syndrome rat model with intermittent hypoxia (IH) exposure as seen in obstructive sleep apnea, on a base of preexisting emphysema caused by 16 wk of smoke exposure to determine whether IH and emphysema existing simultaneously play overlapped roles o...

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Publicado en:Respiratory Care Vol. 60; no. 2; pp. 279 - 290
Autores principales: Qing-chan Yang, Xin Sun, Yong-mei Wang, Qi Wu, Jing Feng, Bao-yuan Chen
Formato: pictorial research tables/charts Journal Article
Publicado: Mary Ann Liebert, Inc. Feb2015
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Feb2015
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      pub: Mary Ann Liebert, Inc.
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        atl: Systematic and Endothelial Inflammation and Endothelial Progenitor Cell Levels in Emphysematous Rats Exposed to Intermittent Hypoxia.
      aug:
        au:
          Qing-chan Yang
          Xin Sun
          Yong-mei Wang
          Qi Wu
          Jing Feng
          Bao-yuan Chen
        affil: Tianjin Haihe Hospital, Tianjin, China.
      sug:
        subj:
          Emphysema Immunology
          Anoxia Immunology
          Endothelium Immunology
          Inflammation
          Sleep Apnea, Obstructive Immunology
          Animal Studies
          Rats
          Descriptive Statistics
          Blood Gas Analysis
          Electroencephalography
          Tumor Necrosis Factor Blood
          Interleukins Blood
          Flow Cytometry
          Funding Source
          Reverse Transcriptase Polymerase Chain Reaction
          Histological Techniques
          Data Analysis Software
          Post Hoc Analysis
          One-Way Analysis of Variance
          Carotid Arteries
      ab: BACKGROUND: This study aimed to develop an overlap syndrome rat model with intermittent hypoxia (IH) exposure as seen in obstructive sleep apnea, on a base of preexisting emphysema caused by 16 wk of smoke exposure to determine whether IH and emphysema existing simultaneously play overlapped roles on systematic/endothelial inflammation and endothelial damage. METHODS: Sixty male Wistar rats were divided into 4 groups of 15 each, labeled according to exposure conditions as control, IH, emphysema, and overlap groups. In these animals, electroencephalogram monitoring and preliminary experiments to obtain arterial blood gas values were performed. Serum concentrations of tumor necrosis factor (TNF)-α and interleukin (IL)-6, TNF-α and IL-6 concentrations in the culture medium, Ras homology A mRNA expression levels of endothelial cells from right common carotid artery, and ratio of carotid intima-media thickness of whole thickness of vascular wall expressed in percent (C-IMT) (%) values were evaluated. Subsequently, circulating endothelial progenitor cells (EPCs) within rat peripheral blood and bone marrow were measured with flow cytometry. RESULTS: The serum and endothelial concentrations of TNF-α and IL-6 and the levels of endothelial Ras homology A mRNA have statistically significant results described as overlap > emphysema > IH > control. The levels of EPCs in rat peripheral blood and bone marrow have statistically significant results described as overlap > IH > emphysema > control. C-IMT (%) values from right common carotid artery are the highest in the overlap group and the lowest in the control group. There is no statistical difference when comparing the IH and the emphysema groups. CONCLUSIONS: Regardless of whether IH and emphysema exposure are mechanistically synergistic, this overlap elicits a more severe systematic/endothelial inflammation and endothelial damage; meanwhile, a robust mobilization of EPCs is demonstrated, which is not to mean a robust adherent and repairing capability.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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