Research. Global Analysis of Posttranscriptional Gene Expression in Response to Sodium Arsenite.

Background: Inorganic arsenic species are potent environmental toxins and causes of numerous health problems. Most studies have assumed that arsenic-induced changes in mRNA levels result from effects on gene transcription. Objectives: We evaluated the prevalence of changes in mRNA stability in respo...

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Publicado en:Environmental Health Perspectives Vol. 123; no. 4; pp. 324 - 331
Autores principales: Lian-Qun Qiu, Abey, Sarah, Harris, Shawn, Shah, Ruchir, Gerrish, Kevin E., Blackshear, Perry J.
Formato: research tables/charts Journal Article
Publicado: National Institute of Environmental Health Sciences Apr2015
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2015
      vid: 123
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      pub: National Institute of Environmental Health Sciences
      place: Research Triangle Park, North Carolina
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        10.1289/ehp.1408626
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        atl: Research. Global Analysis of Posttranscriptional Gene Expression in Response to Sodium Arsenite.
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        au:
          Lian-Qun Qiu
          Abey, Sarah
          Harris, Shawn
          Shah, Ruchir
          Gerrish, Kevin E.
          Blackshear, Perry J.
        affil: Laboratory of Signal Transduction, National Institute of Environmental Health Sciences (NIEHS), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Research Triangle Park, North Carolina, USA
      sug:
        subj:
          Gene Expression
          Arsenates Pharmacodynamics
          Descriptive Statistics
          Data Analysis Software
          Confidence Intervals
          Odds Ratio
          Arsenates Analysis
          Arsenates Adverse Effects
      ab: Background: Inorganic arsenic species are potent environmental toxins and causes of numerous health problems. Most studies have assumed that arsenic-induced changes in mRNA levels result from effects on gene transcription. Objectives: We evaluated the prevalence of changes in mRNA stability in response to sodium arsenite in human fibroblasts. Methods: We used microarray analyses to determine changes in steady-state mRNA levels and mRNA decay rates following 24-hr exposure to noncytotoxic concentrations of sodium arsenite, and we confirmed some of these changes using real-time reverse-transcription polymerase chain reaction (RT-PCR). Results: In arsenite-exposed cells, 186 probe set-identified transcripts were significantly increased and 167 were significantly decreased. When decay rates were analyzed after actinomycin D treatment, only 4,992 (9.1%) of probe set-identified transcripts decayed by > 25% after 4 hr. Of these, 70 were among the 353 whose steady-state levels were altered by arsenite, and of these, only 4 exhibited significantly different decay rates between arsenite and control treatment. Real-time RT-PCR confirmed a major, significant arsenite-induced stabilization of the mRNA encoding δ aminolevulinate synthase 1 (ALAS1), the rate-limiting enzyme in heme biosynthesis. This change presumably accounted for at least part of the 2.7-fold increase in steady-state ALAS1 mRNA levels seen after arsenite treatment. This could reflect decreases in cellular heme caused by the massive induction by arsenite of heme oxygenase mRNA (HMOX1; 68‑fold increase), the rate-limiting enzyme in heme catabolism. Conclusions: We conclude that arsenite modification of mRNA stability is relatively uncommon, but in some instances can result in significant changes in gene expression. Citation: Qiu LQ, Abey S, Harris S, Shah R, Gerrish KE, Blackshear PJ. 2015. Global analysis of posttranscriptional gene expression in response to sodium arsenite. Environ Health Perspect 123:324-330; http://dx.doi.org/10.1289/ehp.1408626
      pubtype: Academic Journal
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      ougenre: Article
    language: English
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