Influence of oral supplementation with sesamin on longevity of Caenorhabditis elegans and the host defense.

Purpose: Nutritional control has been proposed as a potential therapy for slowing the senescence of immune function and decreasing mortality. This study investigated whether sesamin could modify host defense systems and extend the lifespan of the nematode Caenorhabditis elegans. Methods: Nematodes w...

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Publicado en:European Journal of Nutrition Vol. 53; no. 8; pp. 1659 - 1669
Autores principales: Yaguchi, Yukie, Komura, Tomomi, Kashima, Noriko, Tamura, Miho, Kage-Nakadai, Eriko, Saeki, Shigeru, Terao, Keiji, Nishikawa, Yoshikazu
Formato: equations & formulas pictorial research tables/charts Journal Article
Publicado: Springer Nature Dec2014
Acceso en línea:Ver este registro en EBSCOhost
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      jtl: European Journal of Nutrition
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      dt: Dec2014
      vid: 53
      iid: 8
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      pub: Springer Nature
      place: New York, New York
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        99620619
        10.1007/s00394-014-0671-6
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        atl: Influence of oral supplementation with sesamin on longevity of Caenorhabditis elegans and the host defense.
      aug:
        au:
          Yaguchi, Yukie
          Komura, Tomomi
          Kashima, Noriko
          Tamura, Miho
          Kage-Nakadai, Eriko
          Saeki, Shigeru
          Terao, Keiji
          Nishikawa, Yoshikazu
        affil: Department of Food and Human Health Sciences, Graduate School of Human Life Science, Osaka City University, 3-3-138 Sugimoto, Sumiyoshi-ku Osaka 558-8585 Japan
      sug:
        subj:
          Dietary Supplementation
          Longevity Drug Effects
          Immune System Physiology
          Seeds
          Plant Extracts Pharmacodynamics
          Lignans Pharmacodynamics
          Japan
          Funding Source
          In Vivo Studies
          Nematodes
          Movement Evaluation
          Comparative Studies
          Growth Evaluation
          Infection Prevention and Control
          Oxidative Stress
          Models, Statistical
          Kaplan-Meier Estimator
          Log-Rank Test
          T-Tests
          Mann-Whitney U Test
          Kruskal-Wallis Test
          P-Value
          Descriptive Statistics
          Body Size
      ab: Purpose: Nutritional control has been proposed as a potential therapy for slowing the senescence of immune function and decreasing mortality. This study investigated whether sesamin could modify host defense systems and extend the lifespan of the nematode Caenorhabditis elegans. Methods: Nematodes were fed standard food (the bacterium Escherichia coli strain OP50) supplemented with various doses of sesamin/γ-cyclodextrin inclusion compounds starting from young adulthood. The mean lifespan, muscle function, lipofuscin accumulation, protein carbonyl content, and stress resistance of the worms were examined. Then, C. elegans mutants harboring loss-of-function lesions in longevity- and host defense-related signaling pathways were supplemented with sesamin to identify the genes involved in the longevity effects. Results: Worms supplemented with sesamin displayed higher locomotion and prolongevity and produced offspring at levels similar to unsupplemented control animals. The growth curves of nematodes were similar to those of controls, suggesting that sesamin did not induce prolongevity effects through dietary restriction. Notably, sesamin made the worms more resistant to infection by Legionella pneumophila and more resistant to oxidative stressors such as paraquat and hydrogen peroxide and prolonged the lifespan of a mev- 1 mutant that produces abundant superoxide anions. However, the accumulation of protein carbonyls and lipofuscin was similar in sesamin-exposed and control worms, suggesting that sesamin is unlikely to work simply as an antioxidant. Sesamin supplementation failed to extend the lifespan of loss-of-function mutants of daf-2, daf- 16, pmk- 1, and skn- 1. Conclusions: Sesamin enhances the host defense of C. elegans and increases the average lifespan via activation of both skn- 1 (encoding a component of the p38 MAPK pathway) and daf- 16 (encoding a component of the IGF-1 pathway).
      pubtype: Academic Journal
      doctype:
        equations & formulas
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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