The Effect of Left Ventricular Electrical Delay on the Acute Hemodynamic Response with Cardiac Resynchronization Therapy.

Effect of QLV on CRT Hemodynamic Response Introduction Cardiac resynchronization therapy (CRT) improves hemodynamic function, as well as reduces hospitalizations and mortality among patients with systolic dysfunction, QRS prolongation, and heart failure. The magnitude of the hemodynamic response is...

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Publicado en:Journal of Cardiovascular Electrophysiology Vol. 25; no. 6; pp. 624 - 631
Autores principales: Gold, Michael R., Leman, Robert B., Wold, Nicholas, Sturdivant, J. Lacy, Yu, Yinghong
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell Jun2014
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jun2014
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      pub: Wiley-Blackwell
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        atl: The Effect of Left Ventricular Electrical Delay on the Acute Hemodynamic Response with Cardiac Resynchronization Therapy.
      aug:
        au:
          Gold, Michael R.
          Leman, Robert B.
          Wold, Nicholas
          Sturdivant, J. Lacy
          Yu, Yinghong
        affil: Medical University of South Carolina
      sug:
        subj:
          Cardiac Resynchronization Therapy
          Hemodynamics
          Ventricular Dysfunction, Left
          Heart Failure Therapy
          Defibrillators, Implantable Utilization
          Funding Source
          Predictive Value of Tests
          Human
          Middle Age
          Aged
          Male
          Female
          Post Hoc Analysis
          Descriptive Statistics
          Data Analysis Software
          Two-Way Analysis of Variance
          Electrocardiography
          Middle Aged: 45-64 years
          Aged: 65+ years
          Male
          Female
      ab: Effect of QLV on CRT Hemodynamic Response Introduction Cardiac resynchronization therapy (CRT) improves hemodynamic function, as well as reduces hospitalizations and mortality among patients with systolic dysfunction, QRS prolongation, and heart failure. The magnitude of the hemodynamic response is associated with improved outcomes, so optimization of this parameter is a goal of therapy. The purpose of this study was to evaluate the effect of left ventricular (LV) electrical delay, as assessed by the QLV interval, on the acute hemodynamic response to CRT. Methods and Results This study included 31 patients undergoing biventricular ICD placement. At implant, invasive LV dP/dt was measured by a micromanometer catheter during biventricular (BV) or LV only pacing. Both atrial sensing (AS) and atrial pacing (AP) modes were evaluated at 5 different AV delays, tested in randomized order. The QLV interval was measured at the LV pacing site. Compared with intrinsic rhythm, CRT increased LV dP/dtmax by 9.5 ± 8.8% with BV pacing and 10.0 ± 9.2% with LV pacing (P = 0.38) during AS. With AP, CRT increased LV dP/dtmax by 16.0 ± 10.8% and 15.3 ± 11.1%, respectively (P = 0.47). QLV was strongly correlated with the hemodynamic response in all pacing configurations. Multivariate analysis showed that with BV pacing QLV was an independent predictor of the hemodynamic response with a 1.7% increase in %LV dP/dt for every 10 milliseconds prolongation of QLV. Conclusions LV electrical delay is a strong predictor of the acute hemodynamic response to CRT. This relationship is independent of pacing mode.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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