Reduced Latency in the Metastatic Niche Contributes to the More Aggressive Phenotype of LM8 Compared to Dunn Osteosarcoma Cells.

Metastasis is the major cause of death of osteosarcoma patients and its diagnosis remains difficult. In preclinical studies, however, forced expression of reporter genes in osteosarcoma cells has remarkably improved the detection of micrometastases and, consequently, the quality of the studies. We r...

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Publicado en:Sarcoma pp. 1 - 14
Autores principales: Matthias J. E. Arlt, Ingo J. Banke, Josefine Bertz, Ram Mohan Ram Kumar, Roman Muff, Walter Born, Bruno Fuchs
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2013
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2013
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Reduced Latency in the Metastatic Niche Contributes to the More Aggressive Phenotype of LM8 Compared to Dunn Osteosarcoma Cells.
      aug:
        au:
          Matthias J. E. Arlt
          Ingo J. Banke
          Josefine Bertz
          Ram Mohan Ram Kumar
          Roman Muff
          Walter Born
          Bruno Fuchs
        affil: Laboratory for Orthopedic Research, Department of Orthopedics, Balgrist University Hospital, University of Zurich, Forchstrasse 340, 8008 Zurich, Switzerland
      sug:
        subj:
          Osteosarcoma Complications
          Neoplasm Metastasis Pathology
          Phenotype
          Animal Studies
          Mice
          Cell Culture Techniques
          In Vitro Studies
          RNA Analysis
          Microarray Analysis
          Polymerase Chain Reaction
          Blotting, Western
          Kaplan-Meier Estimator
          T-Tests
          Survival Analysis
          Descriptive Statistics
          Gene Expression
          Funding Source
      ab: Metastasis is the major cause of death of osteosarcoma patients and its diagnosis remains difficult. In preclinical studies, however, forced expression of reporter genes in osteosarcoma cells has remarkably improved the detection of micrometastases and, consequently, the quality of the studies. We recently showed that Dunn cells equipped with a lacZ reporter gene disseminated from subcutaneous primary tumors as frequently as their highly metastatic subline LM8, but only LM8 cells grew to macrometastases. In the present time-course study, tail-vein-injected Dunn and LM8 cells settled within 24 h at the same frequency in the lung, liver, and kidney of mice. Furthermore, Dunn cells also grew to macrometastases, but, compared to LM8, with a delay of two weeks in lung and one week in liver and kidney tissue, consistent with prolonged survival of the mice. Dunn- and LM8-cell-derived ovary and spine metastases occurred less frequently. In vitro, Dunn cells showed less invasiveness and stronger contact inhibition and intercellular adhesion than LM8 cells and several cancer- and dormancy-related genes were differentially expressed. In conclusion, Dunn cells, compared to LM8, have a similar capability but a longer latency to form macrometastases and provide an interesting new experimental system to study tumor cell dormancy.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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