Molecular differences between chronic and aggressive periodontitis.

The 2 major forms of periodontitis, chronic (CP) and aggressive (AgP), do not display sufficiently distinct histopathological characteristics or microbiological/immunological features. We used molecular profiling to explore biological differences between CP and AgP and subsequently carried out super...

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Publicado en:Journal of Dental Research Vol. 92; no. 12; pp. 1081 - 1089
Autores principales: Kebschull, M, Guarnieri, P, Demmer, R T, Boulesteix, A L, Pavlidis, P, Papapanou, P N
Formato: research Journal Article
Publicado: Sage Publications Inc. Dec2013
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2013
      vid: 92
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        atl: Molecular differences between chronic and aggressive periodontitis.
      aug:
        au:
          Kebschull, M
          Guarnieri, P
          Demmer, R T
          Boulesteix, A L
          Pavlidis, P
          Papapanou, P N
        affil: Division of Periodontics, Section of Oral and Diagnostic Sciences, Columbia University College of Dental Medicine, New York, NY, USA.
      sug:
        subj:
          Aggressive Periodontitis
          Chronic Periodontitis
          Aggressive Periodontitis Immunology
          Aggressive Periodontitis Pathology
          Algorithms
          Apoptosis
          Pharmacokinetics
          Artificial Intelligence
          Chronic Periodontitis Metabolism
          Chronic Periodontitis Pathology
          Epithelium Pathology
          Gene Expression Profiling Methods
          Gingiva Pathology
          Human
          Microarray Analysis
          Periodontal Attachment Loss
          Periodontal Attachment Loss Pathology
          Periodontics
          Periodontal Pocket
          Periodontal Pocket Pathology
          ROC Curve
          Sensitivity and Specificity
          Signal Transduction
          RNA
          Gene Expression Profiling
      ab: The 2 major forms of periodontitis, chronic (CP) and aggressive (AgP), do not display sufficiently distinct histopathological characteristics or microbiological/immunological features. We used molecular profiling to explore biological differences between CP and AgP and subsequently carried out supervised classification using machine-learning algorithms including an internal validation. We used whole-genome gene expression profiles from 310 'healthy' or 'diseased' gingival tissue biopsies from 120 systemically healthy non-smokers, 65 with CP and 55 with AgP, each contributing with ≥ 2 'diseased' gingival papillae (n = 241; with bleeding-on-probing, probing depth ≥ 4 mm, and clinical attachment loss ≥ 3 mm), and, when available, a 'healthy' papilla (n = 69; no bleeding-on-probing, probing depth ≤ 4 mm, and clinical attachment loss ≤ 4 mm). Our analyses revealed limited differences between the gingival tissue transcriptional profiles of AgP and CP, with genes related to immune responses, apoptosis, and signal transduction overexpressed in AgP, and genes related to epithelial integrity and metabolism overexpressed in CP. Different classifying algorithms discriminated CP from AgP with an area under the curve ranging from 0.63 to 0.99. The small differences in gene expression and the highly variable classifier performance suggest limited dissimilarities between established AgP and CP lesions. Future analyses may facilitate the development of a novel, 'intrinsic' classification of periodontitis based on molecular profiling.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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