Modification of a Rodent Hindlimb Model of Secondary Lymphedema: Surgical Radicality versus Radiotherapeutic Ablation.

Secondary lymphedema is an intractable disease mainly caused by damage of the lymphatic system during surgery, yet studies are limited by the lack of suitable animal models. The purpose of this study was to create an improved model of secondary lymphedema in the hindlimbs of rodents with sustained e...

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Publicado en:BioMed Research International Vol. 2013; pp. 208912 - 208913
Autores principales: Park, Hyung Sub, Jung, In Mok, Choi, Geum Hee, Hahn, Soli, Yoo, Young Sun, Lee, Taeseung
Formato: research Journal Article
Publicado: Wiley-Blackwell 2013
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2013
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Modification of a Rodent Hindlimb Model of Secondary Lymphedema: Surgical Radicality versus Radiotherapeutic Ablation.
      aug:
        au:
          Park, Hyung Sub
          Jung, In Mok
          Choi, Geum Hee
          Hahn, Soli
          Yoo, Young Sun
          Lee, Taeseung
        affil: Department of Surgery, Seoul National University College of Medicine, Seoul 110-799, Republic of Korea ; Department of Surgery, Seoul National University Bundang Hospital, 166 Gumi-ro, Bundang-gu, Seongnam-si, Gyeonggi-do 463-707, Republic of Korea.
      sug:
        subj:
          Fetal Development Physiology
          Lower Extremity Pathology
          Lymphatic System Pathology
          Lymphedema Pathology
          Animal Studies
          Epithelial Cells Pathology
          Epithelial Cells Radiation Effects
          Fetal Development Radiation Effects
          Human
          Lower Extremity Radiation Effects
          Lower Extremity Surgery
          Lymphatic System Radiation Effects
          Male
          Mice
          Models, Biological
          Male
      ab: Secondary lymphedema is an intractable disease mainly caused by damage of the lymphatic system during surgery, yet studies are limited by the lack of suitable animal models. The purpose of this study was to create an improved model of secondary lymphedema in the hindlimbs of rodents with sustained effects and able to mimic human lymphedema. This was achieved by combining previously reported surgical methods and radiation to induce chronic lymphedema. Despite more radical surgical destruction of superficial and deep lymphatic vessels, surgery alone was not enough to sustain increased hindlimb volume. Radiotherapy was necessary to prolong these effects, with decreased lymphatic flow on lymphoscintigraphy, but hindlimb necrosis occurred after 4 weeks due to radiation toxicity. The applicability of this model for studies of therapeutic lymphangiogenesis was subsequently tested by injecting muscle-derived stem cells previously cocultured with the supernatant of human lymphatic endothelial cells in vitro. There was a tendency for increased lymphatic flow which significantly increased lymphatic vessel formation after cell injection, but attenuation of hindlimb volume was not observed. These results suggest that further refinement of the rodent hindlimb model is needed by titration of adequate radiation dosage, while stem cell lymphangiogenesis seems to be a promising approach.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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