Cleavage of BCR-ABL transcripts at the T315I point mutation by DNAzyme promotes apoptotic cell death in imatinib-resistant BCR-ABL leukemic cells.

Detalles Bibliográficos
Publicado en:Leukemia (08876924) Vol. 27; no. 8; pp. 1650 - 1659
Autores principales: Kim, J E, Yoon, S, Choi, B-R, Kim, K P, Cho, Y-H, Jung, W, Kim, D-W, Oh, S, Kim, D-E
Formato: research Journal Article
Publicado: Springer Nature Aug2013
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Aug2013
      vid: 27
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      pub: Springer Nature
      place: Dordrecht, <Blank>
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        2012218136
        10.1038/leu.2013.60
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        atl: Cleavage of BCR-ABL transcripts at the T315I point mutation by DNAzyme promotes apoptotic cell death in imatinib-resistant BCR-ABL leukemic cells.
      aug:
        au:
          Kim, J E
          Yoon, S
          Choi, B-R
          Kim, K P
          Cho, Y-H
          Jung, W
          Kim, D-W
          Oh, S
          Kim, D-E
        affil: Department of Bioscience and Biotechnology, Konkuk University, Seoul Republic of Korea.
      sug:
        subj:
          Apoptosis
          Drug Resistance, Neoplasm
          Enzymes
          Leukemia, Myeloid, Chronic
          Mutation
          Proteins
          Animal Studies
          Apoptosis Drug Effects
          Benzamides Pharmacodynamics
          Cell Line, Tumor
          Cell Physiology
          Chemistry
          Genes
          Heterocyclic Compounds Pharmacodynamics
          Leukemia, Myeloid, Chronic Metabolism
          Mice
          Molecular Structure
          Nucleotides
          Proteins Metabolism
          RNA
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
      ab:
    language: English
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