Neuroprotective Effect of Phosphocreatine on Focal Cerebral Ischemia-Reperfusion Injury.

Phosphocreatine (PCr) is a natural compound, which can donate high-energy phosphate group to ADP to synthesize ATP, even in the absence of oxygen and glucose. At present, it is widely used in cardiac and renal ischemia-reperfusion (IR) disease. In this study, to examine the protective efficacy of PC...

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Publicado en:Journal of Biomedicine & Biotechnology Vol. 2012; pp. 1 - 8
Autores principales: Tiegang Li, Nana Wang, Min Zhao
Formato: equations & formulas pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2012
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2012
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Neuroprotective Effect of Phosphocreatine on Focal Cerebral Ischemia-Reperfusion Injury.
      aug:
        au:
          Tiegang Li
          Nana Wang
          Min Zhao
        affil: Emergency Department, Shengjing Hospital of China Medical University, Shenyang 110004, China
      sug:
        subj:
          Neuroprotective Agents Pharmacodynamics
          Phosphocreatine Pharmacodynamics
          Infarction Drug Therapy
          Reperfusion Injury Drug Therapy
          Treatment Outcomes
          Rats
          Animal Studies
          Descriptive Statistics
          Data Analysis Software
          Repeated Measures
          Analysis of Variance
          Kruskal-Wallis Test
          Apoptosis Drug Effects
          Intracranial Pressure Drug Effects
          Funding Source
      ab: Phosphocreatine (PCr) is a natural compound, which can donate high-energy phosphate group to ADP to synthesize ATP, even in the absence of oxygen and glucose. At present, it is widely used in cardiac and renal ischemia-reperfusion (IR) disease. In this study, to examine the protective efficacy of PCr against cerebral IR, disodium creatine phosphate was injected intravenously into rats before focal cerebral IR. Intracranial pressure (ICP), neurological score, cerebral infarction volume, and apoptotic neurons were observed. Expression of caspase-3 and aquaporin-4 (AQP4) was analyzed. Compared with IR group, rats pretreated with PCr had better neurologic score, less infarction volume, fewer ultrastructural histopathologic changes, reduced apoptosis, and lower aquaporin-4 level. In conclusion, PCr is neuroprotective after transient focal cerebral IR injury. Such a protection might be associated with apoptosis regulating proteins.
      pubtype: Academic Journal
      doctype:
        equations & formulas
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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