Progression of Subclinical Myocardial Dysfunction in Type 2 Diabetes after 5 years Despite Improved Glycemic Control.

Background: Patients with uncomplicated diabetes have reduced left ventricular long-axis function, related to poor glycemic control and increased conduit arterial stiffness, with increased radial function. It is unknown if improved control of risk factors can reverse these subclinical changes. Patie...

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Publicado en:Echocardiography Vol. 29; no. 9; pp. 1045 - 1054
Autores principales: Vintila, Vlad Damian, Roberts, Aled, Vinereanu, Dragos, Fraser, Alan Gordon
Formato: diagnostic images research tables/charts Journal Article
Publicado: Wiley-Blackwell Oct2012
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Oct2012
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      pub: Wiley-Blackwell
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        atl: Progression of Subclinical Myocardial Dysfunction in Type 2 Diabetes after 5 years Despite Improved Glycemic Control.
      aug:
        au:
          Vintila, Vlad Damian
          Roberts, Aled
          Vinereanu, Dragos
          Fraser, Alan Gordon
        affil: University of Medicine and Pharmacy Carol Davila, University and Emergency Hospital of Bucharest, Romania
      sug:
        subj:
          Diabetes Mellitus, Type 2 Complications
          Glycemic Control
          Disease Progression
          Ventricular Dysfunction, Left Etiology
          Myocardial Diseases Etiology
          Human
          Descriptive Statistics
          Male
          Female
          Middle Age
          Echocardiography Methods
          Cross Sectional Studies
          Prospective Studies
          Record Review
          Intrarater Reliability
          Data Analysis Software
          T-Tests
          Chi Square Test
          Logistic Regression
          Funding Source
          Middle Aged: 45-64 years
          Male
          Female
      ab: Background: Patients with uncomplicated diabetes have reduced left ventricular long-axis function, related to poor glycemic control and increased conduit arterial stiffness, with increased radial function. It is unknown if improved control of risk factors can reverse these subclinical changes. Patients and methods: We studied 27 patients with type 2 diabetes (18 men) 57 ± 1 months (mean ± SD) after an initial visit when they were compared with healthy age- and sex-matched controls. On both visits patients had detailed echocardiographic studies including tissue Doppler, noninvasive tests of conduit arterial function, and metabolic and lipid profiling. Results: Mean age at this second review was 63.4 ± 8.1 years; 48% of patients received insulin. Mean HbA1c had decreased by 13% to 8 ± 1.6% and cholesterol by 17% to 4.3 ± 1.3 mmol/L (both, P < 0.01), but long-axis systolic and early diastolic myocardial velocities had also declined, by 13% and 20%, respectively (both, P < 0.001). Body mass index had increased by 4%, arterial pulse pressure by 17% (both P < 0.01), and carotid arterial stiffness by 49% (P < 0.01). Fractional shortening (by 21%, P < 0.001), radial systolic velocity (by 13%, P < 0.05), and ejection fraction (by 9% to 68 ± 7%, P < 0.01) had all declined. Reductions in longitudinal function were best predicted by its baseline measurements, duration of diabetes, fasting triglycerides, and arterial stiffness (epsilon index). Conclusions: Despite improved diabetic control, subclinical left ventricular dysfunction progressed over 5 years. Radial compensation was reversed. Prevention of subclinical myocardial dysfunction in diabetes might require more intensive control of net cardiovascular risk.
      pubtype: Academic Journal
      doctype:
        diagnostic images
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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