Maternal smoking during pregnancy, genetic polymorphisms of metabolic enzymes, and childhood acute leukemia: the ESCALE study (SFCE).

Purpose: This study explored interactions between prenatal exposure to maternal smoking and polymorphisms in metabolic genes in the risk of childhood acute leukemia (AL). Methods: The data were generated by the ESCALE study, which included 764 AL cases and 1,681 controls in 2003-2004. The data on ma...

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Publicado en:Cancer Causes & Control Vol. 23; no. 2; pp. 329 - 346
Autores principales: Bonaventure A, Goujon-Bellec S, Rudant J, Orsi L, Leverger G, Baruchel A, Bertrand Y, Nelken B, Pasquet M, Michel G, Sirvent N, Bordigoni P, Ducassou S, Rialland X, Zelenika D, Hémon D, Clavel J, Bonaventure, Audrey, Goujon-Bellec, Stéphanie, Rudant, Jérémie
Formato: research Journal Article
Publicado: Springer Nature Feb2012
Acceso en línea:Ver este registro en EBSCOhost
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        atl: Maternal smoking during pregnancy, genetic polymorphisms of metabolic enzymes, and childhood acute leukemia: the ESCALE study (SFCE).
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        au:
          Bonaventure A
          Goujon-Bellec S
          Rudant J
          Orsi L
          Leverger G
          Baruchel A
          Bertrand Y
          Nelken B
          Pasquet M
          Michel G
          Sirvent N
          Bordigoni P
          Ducassou S
          Rialland X
          Zelenika D
          Hémon D
          Clavel J
          Bonaventure, Audrey
          Goujon-Bellec, Stéphanie
          Rudant, Jérémie
        affil: Inserm, UMRS 1018, CESP, Team 6 Environmental Epidemiology of Cancer, Villejuif, France
      sug:
        subj:
          Leukemia, Myeloid, Acute
          Prenatal Exposure Delayed Effects
          Smoking
          Adolescence
          Alleles
          Hydrocarbons, Aromatic Metabolism
          Case Control Studies
          Child
          Child, Preschool
          Oxidoreductases
          Hemeproteins
          Hydrolases
          Europe
          Genes
          Female
          Phenotype
          Disease Susceptibility
          Sequence Analysis Methods
          Genotype
          Human
          Leukemia, Myeloid, Acute Metabolism
          Logistic Regression
          Male
          Polymorphism, Genetic
          Pregnancy
          Pregnancy Complications, Neoplastic
          Pregnancy Complications, Neoplastic Metabolism
          Adolescent: 13-18 years
          Child: 6-12 years
          Child, Preschool: 2-5 years
          Female
          Male
      ab: Purpose: This study explored interactions between prenatal exposure to maternal smoking and polymorphisms in metabolic genes in the risk of childhood acute leukemia (AL). Methods: The data were generated by the ESCALE study, which included 764 AL cases and 1,681 controls in 2003-2004. The data on maternal smoking during pregnancy were obtained by standardized telephone interview of the cases' and controls' mothers. The genotypes CYP1A1*2A/2B (rs4646903), CYP2E1*5 (rs2031920, rs3813867), NQO1*2 (rs1800566), NAT2*5 (rs1801280), and EPHX1 exon 3 (rs1051740) and exon 4 (rs2234922) were obtained using a high-throughput platform and imputation for untyped polymorphisms. The analyses were restricted to the 493 cases (433 cases of lymphoblastic (ALL) and 51 of myeloblastic (AML) leukemia) and 441 controls with at least 2 grandparents born in Europe, who were genotyped with individual call rates greater than 95%. Odds ratios were estimated by logistic regression in case-control analyses and, for gene-gene and gene-environment interactions, by case-only analyses. Results: ALL and AML were not associated with either maternal smoking during pregnancy or candidate polymorphisms in CYP1A1, CYP2E1, EPHX1, and NQO1. Carrying two NAT2*5 alleles was significantly associated with ALL (OR = 1.8 [1.3-2.5]). The analyses also suggested an interaction between three genes involved in benzene metabolism CYP2E1, NQO1, and EPHX1. There was no interaction between maternal smoking and any of the polymorphisms under study. Conclusions: The ESCALE study did not evidence the interaction between CYP1A1*2A/2B and maternal smoking suggested previously. The association with NAT2*5 and the gene-gene interactions need to be replicated.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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