Gastrointestinal symptoms in type-1 diabetes: Is it all about brain plasticity?

BACKGROUND AND AIMS: Autonomic neuropathy seems to play a central role in the development of gastrointestinal symptoms in diabetes. In order to explore the neuronal mechanisms behind the symptoms we evaluated the brain processing of painful visceral stimuli. METHODS: Evoked brain potentials were rec...

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Publicado en:European Journal of Pain Vol. 15; no. 3; pp. 249 - 258
Autores principales: Frøkjær JB, Egsgaard LL, Graversen C, Søfteland E, Dimcevski G, Blauenfeldt RA, Drewes AM
Formato: research Journal Article
Publicado: Wiley-Blackwell Mar2011
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Mar2011
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      pub: Wiley-Blackwell
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        atl: Gastrointestinal symptoms in type-1 diabetes: Is it all about brain plasticity?
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          Frøkjær JB
          Egsgaard LL
          Graversen C
          Søfteland E
          Dimcevski G
          Blauenfeldt RA
          Drewes AM
        affil: Mech-Sense, Department of Gastroenterology, Aalborg Hospital, Aarhus University Hospital, Aalborg, Denmark; Department of Radiology, Aalborg Hospital, Aarhus University Hospital, Aalborg, Denmark.
      sug:
        subj:
          Brain Physiopathology
          Diabetes Mellitus, Type 1 Physiopathology
          Esophagus Physiopathology
          Evoked Potentials Physiology
          Pain Physiopathology
          Adult
          Analysis of Variance
          Autonomic Nervous System Diseases Diagnosis
          Autonomic Nervous System Diseases Physiopathology
          Blood Glucose
          Diabetic Neuropathies Diagnosis
          Diabetic Neuropathies Physiopathology
          Electric Stimulation
          Electroencephalography
          Female
          Gastrointestinal Diseases Diagnosis
          Gastrointestinal Diseases Physiopathology
          Male
          Middle Age
          Adult: 19-44 years
          Middle Aged: 45-64 years
          Female
          Male
      ab: BACKGROUND AND AIMS: Autonomic neuropathy seems to play a central role in the development of gastrointestinal symptoms in diabetes. In order to explore the neuronal mechanisms behind the symptoms we evaluated the brain processing of painful visceral stimuli. METHODS: Evoked brain potentials were recorded to assess the response to painful oesophageal electrical stimuli in 15 healthy volunteers and 14 type-1 diabetes patients with autonomic neuropathy and related gastrointestinal symptoms. Source reconstruction analysis (fixed Multiple Signal Classification (MUSIC) algorithm) was applied to estimate the location of the evoked electrical activity in the brain. RESULTS: The patients had increased oesophageal sensory thresholds compared to the controls (P=0.004). The latencies of the evoked brain potentials at vertex (Cz) were increased (P=0.007) and amplitudes reduced (P=0.011) in diabetics. Compared with controls the patients had a posterior shift of the electrical sources in the anterior cingulate cortex at 54ms, and additional sources close to the posterior insula at 95ms and in medial frontal gyrus at 184ms. CONCLUSIONS: There is evidence of altered central processing to visceral stimulation, and both peripheral and central mechanisms seem involved. Central neuronal reorganisation may contribute to our understanding of the gastrointestinal symptoms in patients with diabetic autonomic neuropathy and this may guide development and evaluation of new treatment modalities.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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