Using Perceptual Signatures to Define and Dissociate Condition-Specific Neural Etiology: Autism and Fragile X Syndrome as Model Conditions.

The functional link between genetic alteration and behavioral end-state is rarely straightforward and never linear. Cases where neurodevlopmental conditions defined by a distinct genetic etiology share behavioral phenotypes are exemplary, as is the case for autism and Fragile X Syndrome (FXS). In th...

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Publicado en:Journal of Autism & Developmental Disorders Vol. 40; no. 12; pp. 1531 - 1541
Autores principales: Bertone A, Hanck J, Kogan C, Chaudhuri A, Cornish K
Formato: tables/charts Journal Article
Publicado: Springer Nature Dec2010
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2010
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      pub: Springer Nature
      place: New York, New York
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        atl: Using Perceptual Signatures to Define and Dissociate Condition-Specific Neural Etiology: Autism and Fragile X Syndrome as Model Conditions.
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          Bertone A
          Hanck J
          Kogan C
          Chaudhuri A
          Cornish K
        affil: McGill Child Laboratory for Research and Education in Developmental Disorders, McGill University, Montreal Canada
      sug:
        subj:
          Autism Spectrum Disorder Physiopathology
          Fragile X Syndrome Physiopathology
          Genotype
          Neural Pathways
          Autism Spectrum Disorder Familial and Genetic
          Fragile X Syndrome Familial and Genetic
          Conceptual Framework
      ab: The functional link between genetic alteration and behavioral end-state is rarely straightforward and never linear. Cases where neurodevlopmental conditions defined by a distinct genetic etiology share behavioral phenotypes are exemplary, as is the case for autism and Fragile X Syndrome (FXS). In this paper and its companion paper, we propose a method for assessing the functional link between genotype and neural alteration across these target conditions by comparing their perceptual signatures. In the present paper, we discuss how such signatures can be used to (1) define and differentiate various aspects of neural functioning in autism and FXS, and subsequently, (2) to infer candidate causal (genetic) mechanisms based on such signatures (see companion paper, this issue).
      pubtype: Academic Journal
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        Journal Article
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    language: English
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