Endocytosis and recycling of tight junction proteins in inflammation.

A critical function of the epithelial lining is to form a barrier that separates luminal contents from the underlying interstitium. This barrier function is primarily regulated by the apical junctional complex (AJC) consisting of tight junctions (TJs) and adherens junctions (AJs) and is compromised...

Descripción completa

Detalles Bibliográficos
Publicado en:Journal of Biomedicine & Biotechnology pp. 484987 - 484988
Autores principales: Utech M, Mennigen R, Bruewer M
Formato: Journal Article
Publicado: Wiley-Blackwell 2010
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=105082000&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 105082000
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        11107243
        137K
      jtl: Journal of Biomedicine & Biotechnology
      issn: 11107243
      maglogo: N
    pubinfo:
      dt: 2010
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
    artinfo:
      ui:
        105082000
        2010748336
        NLM20011071
        PMC2789582
        105082000
      ppf: 484987
      ppct: 1
      formats:
        fmt:
          @attributes:
            type: P
      tig:
        atl: Endocytosis and recycling of tight junction proteins in inflammation.
      aug:
        au:
          Utech M
          Mennigen R
          Bruewer M
        affil: Department of General and Visceral Surgery, University Hospital Muenster, 48149 Muenster, Germany.
      sug:
        subj:
          Endocytosis Physiology
          Inflammation Metabolism
          Membrane Proteins Metabolism
          Cell Membrane Metabolism
          Animals
          Intestinal Mucosa
          Intestinal Mucosa Metabolism
      ab: A critical function of the epithelial lining is to form a barrier that separates luminal contents from the underlying interstitium. This barrier function is primarily regulated by the apical junctional complex (AJC) consisting of tight junctions (TJs) and adherens junctions (AJs) and is compromised under inflammatory conditions. In intestinal epithelial cells, proinflammatory cytokines, for example, interferon-gamma (IFN-gamma), induce internalization of TJ proteins by endocytosis. Endocytosed TJ proteins are passed into early and recycling endosomes, suggesting the involvement of recycling of internalized TJ proteins. This review summarizes mechanisms by which TJ proteins under inflammatory conditions are internalized in intestinal epithelial cells and point out comparable mechanism in nonintestinal epithelial cells.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N