Detection of fetomaternal genotype associations in early-onset disorders: evaluation of different methods and their application to childhood leukemia.

Several designs and analytical approaches have been proposed to dissect offspring from maternal genetic contributions to early-onset diseases. However, lack of parental controls halts the direct verification of the assumption of mating symmetry (MS) required to assess maternally-mediated effects. In...

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Publicado en:Journal of Biomedicine & Biotechnology pp. 13p - 14
Autores principales: Healy J, Bourgey M, Richer C, Sinnett D, Roy-Gagnon MH
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2010
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2010
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2010/369534
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        atl: Detection of fetomaternal genotype associations in early-onset disorders: evaluation of different methods and their application to childhood leukemia.
      aug:
        au:
          Healy J
          Bourgey M
          Richer C
          Sinnett D
          Roy-Gagnon MH
        affil: Sainte-Justine Hospital Research Center, University of Montreal, Montreal, QC, Canada H3T 1C5.
      sug:
        subj:
          Genotype In Utero
          Leukemia In Infancy and Childhood
          Chi Square Test
          Child
          Fetus
          Fisher's Exact Test
          Funding Source
          Human
          Logistic Regression
          Simulations
          Child: 6-12 years
          Fetus, conception to birth
      ab: Several designs and analytical approaches have been proposed to dissect offspring from maternal genetic contributions to early-onset diseases. However, lack of parental controls halts the direct verification of the assumption of mating symmetry (MS) required to assess maternally-mediated effects. In this study, we used simulations to investigate the performance of existing methods under mating asymmetry (MA) when parents of controls are missing. Our results show that the log-linear, likelihood-based framework using a case-triad/case-control hybrid design provides valid tests for maternal genetic effects even under MA. Using this approach, we examined fetomaternal associations between 29 SNPs in 12 cell-cycle genes and childhood pre-B acute lymphoblastic leukemia (ALL). We identified putative fetomaternal effects at loci CDKN2A rs36228834 (P = .017) and CDKN2B rs36229158 (P = .022) that modulate the risk of childhood ALL. These data further corroborate the importance of the mother's genotype on the susceptibility to early-onset diseases.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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