Ultrasound accelerates healing of normal wounds but not of ischemic ones.

To examine the influence of therapeutic ultrasound (US) on repair of standard and ischemic cutaneous lesions, full-thickness excisional wounds were made in rats and treated with a US 3 MHz, 0.5 W/cm2 pulsed duty cycle. We used five experimental groups: control (received US powered off on the day of...

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Published in:Wound Repair & Regeneration Vol. 17; no. 6; pp. 825 - 832
Main Authors: Altomare M, Nascimento AP, Romana-Souza B, Amadeu TP, Monte-Alto-Costa A
Format: pictorial research tables/charts Journal Article
Published: Wiley-Blackwell Nov/Dec2009
Online Access:View this record in EBSCOhost
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        10671927
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      jtl: Wound Repair & Regeneration
      issn: 10671927
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    pubinfo:
      dt: Nov/Dec2009
      vid: 17
      iid: 6
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        105337195
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        2010458067
        10.1111/j.1524-475x.2009.00542.x
        105337195
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      tig:
        atl: Ultrasound accelerates healing of normal wounds but not of ischemic ones.
      aug:
        au:
          Altomare M
          Nascimento AP
          Romana-Souza B
          Amadeu TP
          Monte-Alto-Costa A
        affil: Histology and Embryology Department, State University of Rio de Janeiro, Rio de Janeiro, Brazil
      sug:
        subj:
          Ischemia Complications
          Ultrasonography Methods
          Wound Care Methods
          Wound Healing Physiology
          Animal Studies
          Descriptive Statistics
          Immunohistochemistry
          Models, Biological
          Rats
      ab: To examine the influence of therapeutic ultrasound (US) on repair of standard and ischemic cutaneous lesions, full-thickness excisional wounds were made in rats and treated with a US 3 MHz, 0.5 W/cm2 pulsed duty cycle. We used five experimental groups: control (received US powered off on the day of surgery, and on the second and fourth day), control US (received US on the day of surgery, and on the second and fourth day), ischemic (received US powered off on the day of surgery, and on the second and fourth day), ischemic US 3X (received US on the day of surgery, and on the second and fourth day) and ischemic US 5X (received US in the day of surgery, first, second, third and fourth day). The control US group showed acceleration in wound contraction 7 days after wounding, an increase in collagen density, and only focal inflammatory areas. Neo-epidermis formation was more advanced in the control US group than in the control one. Wound contraction was delayed in the ischemic group when compared with the control group as well as the ischemic US 3X group, was but slightly accelerated in the ischemic US 5X group when compared with the ischemic group 7 days after wounding. Reepithelialization was delayed in both ischemic US groups when compared with the ischemic group. The number of inflammatory cells was higher in both US ischemic groups. We conclude that US therapy accelerates wound healing in normal wounds and delays wound healing in ischemic wounds.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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