Genomewide analysis of aryl hydrocarbon receptor binding targets reveals an extensive array of gene clusters that control morphogenetic and developmental programs.

Background: The vertebrate aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor that regulates cellular responses to environmental polycyclic and halogenated compounds. The naive receptor is believed to reside in an inactive cytosolic complex that translocates to the nucleus an...

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Publicado en:Environmental Health Perspectives Vol. 117; no. 7; pp. 1139 - 1147
Autores principales: Sartor MA, Schnekenburger M, Marlowe JL, Reichard JF, Wang Y, Fan Y, Ma C, Karyala S, Halbleib D, Liu X, Medvedovic M, Puga A
Formato: pictorial research tables/charts Journal Article
Publicado: National Institute of Environmental Health Sciences Jul2009
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul2009
      vid: 117
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      pub: National Institute of Environmental Health Sciences
      place: Research Triangle Park, North Carolina
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        10.1289/ehp.0800485
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        atl: Genomewide analysis of aryl hydrocarbon receptor binding targets reveals an extensive array of gene clusters that control morphogenetic and developmental programs.
      aug:
        au:
          Sartor MA
          Schnekenburger M
          Marlowe JL
          Reichard JF
          Wang Y
          Fan Y
          Ma C
          Karyala S
          Halbleib D
          Liu X
          Medvedovic M
          Puga A
        affil: Laboratory for Statistical Genomics and Systems Biology, Department of Environmental Health and Center for Environmental Genetics, University of Cincinnati College of Medicine, Cincinnati, Ohio, USA
      sug:
        subj:
          Gene Expression Drug Effects
          Hydrocarbons
          Animal Studies
          Funding Source
          Mice
          Reverse Transcriptase Polymerase Chain Reaction
      ab: Background: The vertebrate aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor that regulates cellular responses to environmental polycyclic and halogenated compounds. The naive receptor is believed to reside in an inactive cytosolic complex that translocates to the nucleus and induces transcription of xenobiotic detoxification genes after activation by ligand. Objectives: We conducted an integrative genomewide analysis of AHR gene targets in mouse hepatoma cells and determined whether AHR regulatory functions may take place in the absence of an exogenous ligand. Methods: The network of AHR-binding targets in the mouse genome was mapped through a multipronged approach involving chromatin immunoprecipitation/chip and global gene expression signatures. The findings were integrated into a prior functional knowledge base from Gene Ontology, interaction networks, Kyoto Encyclopedia of Genes and Genomes pathways, sequence motif analysis, and literature molecular concepts. Results: We found the naive receptor in unstimulated cells bound to an extensive array of gene clusters with functions in regulation of gene expression, differentiation, and pattern specification, connecting multiple morphogenetic and developmental programs. Activation by the ligand displaced the receptor from some of these targets toward sites in the promoters of xenobiotic metabolism genes. Conclusions: The vertebrate AHR appears to possess unsuspected regulatory functions that may be potential targets of environmental injury.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
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      ougenre: Article
    language: English
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