Alterations of the cell cycle regulators cyclin D1, cyclin A, p27, p21, p16, and pRb in apocrine metaplasia of the breast.

G1/S transition defects have been a proposed requirement for tumor development. Apocrine metaplasia (APM) in the breast has been held as a sign of benignity. Yet, a number of studies have reported the presence of molecular abnormalities in some forms of APM suggesting a possible oncogenic potential...

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Published in:Breast Journal Vol. 15; no. 5; pp. 475 - 483
Main Authors: Elayat G, Selim AA, Wells CA
Format: pictorial research tables/charts Journal Article
Published: Wiley-Blackwell Sep/Oct2009
Online Access:View this record in EBSCOhost
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      dt: Sep/Oct2009
      vid: 15
      iid: 5
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        2010385354
        10.1111/j.1524-4741.2009.00762.x
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        atl: Alterations of the cell cycle regulators cyclin D1, cyclin A, p27, p21, p16, and pRb in apocrine metaplasia of the breast.
      aug:
        au:
          Elayat G
          Selim AA
          Wells CA
        affil: Department of Histopathology, St Bartholomew's Hospital, West Smithfield, London, United Kingdom
      sug:
        subj:
          Antibodies
          Breast Neoplasms
          Cells
          Immunohistochemistry
          Adult
          Aged
          Aged, 80 and Over
          Chi Square Test
          Descriptive Statistics
          Female
          Fisher's Exact Test
          Funding Source
          Mann-Whitney U Test
          Middle Age
          P-Value
          United Kingdom
          Human
          Adult: 19-44 years
          Aged: 65+ years
          Aged, 80 & over
          Middle Aged: 45-64 years
          Female
      ab: G1/S transition defects have been a proposed requirement for tumor development. Apocrine metaplasia (APM) in the breast has been held as a sign of benignity. Yet, a number of studies have reported the presence of molecular abnormalities in some forms of APM suggesting a possible oncogenic potential for some of these lesions. We currently investigate the role of some of the cell cycle proteins, previously reported to be de-regulated in breast cancer, in an attempt to assess their significance in APM. Using immunohistochemistry, the expression of cyclin D1, cyclin A, p27, p21, p16, pRb and Ki-67 was examined in 93 cases of APM. The cases were divided into nonpapillary (NAPM) (30 cases) and papillary metaplasia (PAPM) (63 cases). PAPM was further subdivided into simple papillary (SPAPM) (29 cases), complex papillary (28 cases) and highly complex papillary (six cases). For statistical analysis, the last two groups were merged together (CPAPM). The results showed that increased histological complexity was associated with significant increase of proliferative capacity and alterations of the cell cycle control. The median Ki-67 index was 1.5% in SPAPM and 4.8% in the CPAPM. Also, alterations of the cell cycle regulators were significantly higher in the CPAPM than in the SPAPM. NAPM was generally similar to normal breast epithelium. Apocrine cells were negative for p16 while pRb was expressed in all cases. These findings suggest that in complex forms of APM, there is a considerable degree of cellular unrest. This may contribute to increased susceptibility to carcinogenesis.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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