Biphasic regulation of cell death and survival by hydrophobic bile acids in HCT116 cells.

A secondary bile acid, namely, deoxycholic acid (DCA), has been known to promote colon tumors; on the other hand, it also induces apoptosis in several human colon cancer cell lines. A hydrophobic primary bile acid, namely, chenodeoxycholic acid (CDCA), exhibits a similar property of apoptosis induct...

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Publicado en:Nutrition & Cancer Vol. 61; no. 3; pp. 374 - 381
Autores principales: Yui S, Kanamoto R, Saeki T
Formato: research tables/charts Journal Article
Publicado: Taylor & Francis Ltd May/Jun2009
Acceso en línea:Ver este registro en EBSCOhost
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      dt: May/Jun2009
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      pub: Taylor & Francis Ltd
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        atl: Biphasic regulation of cell death and survival by hydrophobic bile acids in HCT116 cells.
      aug:
        au:
          Yui S
          Kanamoto R
          Saeki T
        affil: Laboratory of Molecular Nutrition, Graduate School of Life and Environmental Sciences, Kyoto Prefectural University, Kyoto 606-8522, Japan.
      sug:
        subj:
          Bile Acids and Salts
          Cell Death Evaluation
          Cell Physiology
          Survival
          Analysis of Variance
          Blotting, Western
          Neoplasms Prevention and Control
          Nutrition
          Post Hoc Analysis
          Human
      ab: A secondary bile acid, namely, deoxycholic acid (DCA), has been known to promote colon tumors; on the other hand, it also induces apoptosis in several human colon cancer cell lines. A hydrophobic primary bile acid, namely, chenodeoxycholic acid (CDCA), exhibits a similar property of apoptosis induction; DCA and CDCA also trigger some specific intracellular signal pathways in the human colon cancer cell line HCT116. In this article, we report that hydrophobic bile acids induce different cellular responses depending on their concentration, that is, a sublethal concentration of hydrophobic bile acids can suppress the apoptosis induced by a higher concentration of DCA. Pretreatment with DCA or CDCA at a concentration of < or = 200 microM for 8 h suppressed the apoptosis induced by 500 microM DCA in HCT116 cells. Under this condition, the association of caspase-9 and Apaf-1 and subsequent activation of caspase-9 were inhibited, but the release of cytochrome c from the mitochondria was not. At 200 microM, DCA and CDCA induced the phosphorylation of Akt and ERK1/2, although these phosphorylations do not appear to be indispensable for the cytoprotection. It is interpreted that prolonged exposure to sublethal concentrations of hydrophobic bile acids induces resistance to apoptosis, leading to promotion of colorectal tumorigenesis.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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