Gene expression profiling in Brodmann's area 46 from subjects with schizophrenia.

OBJECTIVE: To identify altered gene expression in the dorsolateral prefrontal cortex obtained after death from subjects with schizophrenia. METHOD: Restriction fragment differential display (RFDD) was used to measure levels of mRNA in Brodmann area (BA) 46 from schizophrenia and control subjects. Fi...

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Publicado en:Australian & New Zealand Journal of Psychiatry Vol. 41; no. 4; pp. 308 - 321
Autores principales: Dean B, Keriakous D, Scarr E, Thomas EA
Formato: Journal Article
Publicado: Sage Publications Inc. Apr2007
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2007
      vid: 41
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      pub: Sage Publications Inc.
      place: Thousand Oaks, California
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        atl: Gene expression profiling in Brodmann's area 46 from subjects with schizophrenia.
      aug:
        au:
          Dean B
          Keriakous D
          Scarr E
          Thomas EA
        affil: Rebecca L. Cooper Research Laboratories, Mental Health Research Institute of Victoria, Locked Bag 11, Parkville, Vic. 3052, Australia. bdean_mhri@iprimus.com.au
      sug:
        subj:
          Frontal Lobe Metabolism
          Frontal Lobe Physiopathology
          Genetic Techniques Methods
          Proteins Metabolism
          Proteins
          Schizophrenia Metabolism
          Schizophrenia Physiopathology
          Schizophrenia
          Adult
          Animals
          Bipolar Disorder Metabolism
          Bipolar Disorder Physiopathology
          Bipolar Disorder
          DNA
          Female
          Male
          Nerve Tissue Proteins Metabolism
          Nerve Tissue Proteins
          Polymerase Chain Reaction
          Polymorphism, Genetic
          Rats
          RNA
          Signal Transduction
          Adult: 19-44 years
          Female
          Male
      ab: OBJECTIVE: To identify altered gene expression in the dorsolateral prefrontal cortex obtained after death from subjects with schizophrenia. METHOD: Restriction fragment differential display (RFDD) was used to measure levels of mRNA in Brodmann area (BA) 46 from schizophrenia and control subjects. Findings on specific mRNA identified with RFDD were further investigated using real-time polymerase chain reaction (real-time PCR), PCR and western blotting. RESULTS: Levels of mRNA for 63 of approximately 12,500 genes differed in BA 46 in schizophrenia. Subsequent real-time PCR has shown that mRNA for muscleblind protein 1 (MBNL1) and protocadherin 17 (PCDH17) are increased in BA 46 from subjects with schizophrenia of short, but not long, duration. Altered levels of mRNA for neither gene were present in BA 9 from subjects with schizophrenia or in either cortical area from subjects with bipolar 1 disorder. By contrast, both RFDD and real-time PCR failed to show altered expression of the schizophrenia candidate gene disrupted in schizophrenia 1 (DISC1) BA46 from any diagnostic cohort. CONCLUSION: The present study has identified genes that are differentially expressed in BA 46 in schizophrenia. Initial studies have shown that there is a need for a careful validation of genes shown to be affected in schizophrenia using high-throughput technologies. In addition the present study has shown that gene expression may vary considerably depending on the duration of schizophrenia. This raises the hypothesis that changing gene expression may be underlying the change in symptom profile that occurs with disease progression in some subjects with schizophrenia.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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