Diesel exhaust inhalation elicits acute vasoconstriction in vivo.

BACKGROUND: Traffic-related air pollution is consistently associated with cardiovascular morbidity and mortality. Recent human and animal studies suggest that exposure to air pollutants affects vascular function. Diesel exhaust (DE) is a major source of traffic-related air pollution. OBJECTIVES: Our...

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Publicado en:Environmental Health Perspectives Vol. 116; no. 7; pp. 937 - 943
Autores principales: Peretz A, Sullivan JH, Leotta DF, Trenga CA, Sands FN, Allen J, Carlsten C, Wilkinson CW, Gill EA, Kaufman JD
Formato: clinical trial research Journal Article
Publicado: National Institute of Environmental Health Sciences Jul2008
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul2008
      vid: 116
      iid: 7
      pid: 56539
      pub: National Institute of Environmental Health Sciences
      place: Research Triangle Park, North Carolina
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        105654683
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        10.1289/ehp.11027
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        atl: Diesel exhaust inhalation elicits acute vasoconstriction in vivo.
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        au:
          Peretz A
          Sullivan JH
          Leotta DF
          Trenga CA
          Sands FN
          Allen J
          Carlsten C
          Wilkinson CW
          Gill EA
          Kaufman JD
        affil: Occupational and Environmental Medicine Program, Department of Environmental and Occupational Health Sciences, University of Washington, Seattle, Washington, USA
      sug:
        subj:
          Air Pollution
          Brachial Artery Drug Effects
          Environmental Exposure Adverse Effects
          Metabolic Syndrome X Physiopathology
          Particulate Matter
          Vasoconstriction Drug Effects
          Adult
          Brachial Artery Physiopathology
          Brachial Artery Ultrasonography
          Catecholamines Metabolism
          Crossover Design
          Dose-Response Relationship, Drug
          Double-Blind Studies
          Endothelins Metabolism
          Female
          Male
          Metabolic Syndrome X Metabolism
          Middle Age
          Time Factors
          Vasodilation Drug Effects
          Human
          Adult: 19-44 years
          Middle Aged: 45-64 years
          Female
          Male
      ab: BACKGROUND: Traffic-related air pollution is consistently associated with cardiovascular morbidity and mortality. Recent human and animal studies suggest that exposure to air pollutants affects vascular function. Diesel exhaust (DE) is a major source of traffic-related air pollution. OBJECTIVES: Our goal was to study the effects of short-term exposure to DE on vascular reactivity and on mediators of vascular tone. METHODS: In a double-blind, crossover, controlled exposure study, 27 adult volunteers (10 healthy and 17 with metabolic syndrome) were exposed in randomized order to filtered air (FA) and each of two levels of diluted DE (100 or 200 microg/m(3) of fine particulate matter) in 2-hr sessions. Before and after each exposure, we assessed the brachial artery diameter (BAd) by B-mode ultrasound and collected blood samples for endothelin-1 (ET-1) and catecholamines. Postexposure we also assessed endothelium-dependent flow-mediated dilation (FMD). RESULTS: Compared with FA, DE at 200 microg/m(3) elicited a decrease in BAd (0.11 mm; 95% confidence interval, 0.02-0.18), and the effect appeared linearly dose related with a smaller effect at 100 microg/m(3). Plasma levels of ET-1 increased after 200 microg/m(3) DE but not after FA (p = 0.01). There was no consistent impact of DE on plasma catecholamines or FMD. CONCLUSIONS: These results demonstrate that short-term exposure to DE is associated with acute endothelial response and vasoconstriction of a conductance artery. Elucidation of the signaling pathways controlling vascular tone that underlie this observation requires further study.
      pubtype: Academic Journal
      doctype:
        clinical trial
        research
        Journal Article
      ougenre: Article
    language: English
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