Avoidance of right ventricular pacing in cardiac resynchronization therapy improves right ventricular hemodynamics in heart failure patients.

BACKGROUND: Cardiac resynchronization therapy (CRT) applied by pacing the left and right ventricles (BiV) has been shown to provide synchronous left ventricular (LV) contraction in heart failure patients. CRT may also be accomplished through synchronization of a properly timed LV pacing impulse with...

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Publicado en:Journal of Cardiovascular Electrophysiology Vol. 18; no. 5; pp. 497 - 505
Autores principales: Lee KL, Burnes JE, Mullen TJ, Hettrick DA, Tse HF, Lau CP
Formato: Journal Article
Publicado: Wiley-Blackwell May2007
Acceso en línea:Ver este registro en EBSCOhost
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        atl: Avoidance of right ventricular pacing in cardiac resynchronization therapy improves right ventricular hemodynamics in heart failure patients.
      aug:
        au:
          Lee KL
          Burnes JE
          Mullen TJ
          Hettrick DA
          Tse HF
          Lau CP
      sug:
        subj:
          Cardiac Resynchronization Therapy Methods
          Heart Failure Prevention and Control
          Ventricular Dysfunction, Left Prevention and Control
          Ventricular Dysfunction, Right Prevention and Control
          Female
          Heart Failure Complications
          Male
          Middle Age
          Treatment Outcomes
          Ventricular Dysfunction, Left Etiology
          Ventricular Dysfunction, Right Etiology
          Middle Aged: 45-64 years
          Female
          Male
      ab: BACKGROUND: Cardiac resynchronization therapy (CRT) applied by pacing the left and right ventricles (BiV) has been shown to provide synchronous left ventricular (LV) contraction in heart failure patients. CRT may also be accomplished through synchronization of a properly timed LV pacing impulse with intrinsically conducted activation wave fronts. Elimination of right ventricular (RV) pacing may provide a more physiological RV contraction pattern and reduce device current drain. We evaluated the effects of LV and BiV pacing over a range of atrioventricular intervals on the performance of both ventricles. METHODS: Acute LV and RV hemodynamic data from 17 patients with heart failure (EF = 30 +/- 1%) and a wide QRS (138 +/- 25 msec) or mechanical dyssynchrony were acquired during intrinsic rhythm, BiV, and LV pacing. RESULTS: The highest LV dP/dt(max) was achieved during LV pre- (LV paced prior to an RV sense) and BiV pacing, followed by that obtained during LV post-pacing (LV paced after an RV sense) and the lowest LV dP/dt(max) was recorded during intrinsic rhythm. Compared with BiV pacing, LV pre-pacing significantly improved RV dP/dt(max) (378 +/- 136 mmHg/second vs 397 +/- 136 mmHg/second, P < 0.05) and preserved RV cycle efficiency (61.6 +/- 14.6% vs 68.6 +/- 11.4%, P < 0.05) and stroke volume (6.6 +/- 4.4 mL vs 9.0 +/- 6.3 mL, P < 0.05). Based on LV dP/dt(max), the optimal atrioventricular interval could be estimated by subtracting 30 msec from the intrinsic atrial to sensed RV interval. CONCLUSIONS: Synchronized LV pacing produces acute LV and systemic hemodynamic benefits similar to BiV pacing. LV pacing at an appropriate atrioventricular interval prior to the RV sensed impulse provides superior RV hemodynamics compared with BiV pacing.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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