Hyperoxia-induced changes in human airway epithelial cells: the protective effect of perflubron.

OBJECTIVE: To determine the protective effect of perflubron (PFB), a type of perfluorochemical liquid, in hyperoxia-induced cellular injury in the human airway epithelial cells. DESIGN: A controlled, in vitro laboratory study. SETTING: Tertiary-care children's hospital. SUBJECTS: Human airway epithe...

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Published in:Pediatric Critical Care Medicine Vol. 6; no. 2; pp. 188 - 195
Main Authors: Babu PBR, Chidekel A, Shaffer TH
Format: pictorial research tables/charts Journal Article
Published: Lippincott Williams & Wilkins 2005 Mar
Online Access:View this record in EBSCOhost
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      dt: 2005 Mar
      vid: 6
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      pub: Lippincott Williams & Wilkins
      place: Baltimore, Maryland
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        10.1097/01.pcc.0000154944.67042.4f
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        atl: Hyperoxia-induced changes in human airway epithelial cells: the protective effect of perflubron.
      aug:
        au:
          Babu PBR
          Chidekel A
          Shaffer TH
        affil: Nemours Research Lung Center, Alfred I. duPont Hospital for Children, Wilmington, DE
      sug:
        subj:
          Antiobesity Agents Therapeutic Use
          Hyperoxia
          Analysis of Variance
          Funding Source
          Hospitals, Pediatric
          In Vitro Studies
          Repeated Measures
          Human
      ab: OBJECTIVE: To determine the protective effect of perflubron (PFB), a type of perfluorochemical liquid, in hyperoxia-induced cellular injury in the human airway epithelial cells. DESIGN: A controlled, in vitro laboratory study. SETTING: Tertiary-care children's hospital. SUBJECTS: Human airway epithelial cells. INTERVENTIONS: Human airway epithelial cells, Calu-3 cells, grown on polycarbonate porous filters at an air-liquid interface culture were exposed to normoxic (Fico(2) = 5%, balance air) or hyperoxic (Fio(2) = 95%, balance CO(2)) conditions. Hyperoxia-induced cellular changes were monitored by measuring transepithelial resistance (TER) of monolayers, histology of cells, total protein, and interleukin-8 (IL-8) secretion in apical surface fluid (ASF) washings. Under hyperoxic conditions, the protective effect of PFB was assessed by directly adding PFB liquid to the apical surface of monolayers. MEASUREMENTS AND MAIN RESULTS: During hyperoxic gas-liquid interface culture, Calu-3 monolayers exhibited a loss of cellular integrity morphologically, decreased protein concentration, and IL-8 level in ASF washings. During hyperoxic PFB-liquid interface culture, there was an overall increase in TER value of monolayers, improved histology, decreased total protein secretion in ASF washings, and unaltered IL-8 secretion. Cytomorphologic observations of PFB-treated Calu-3 cells indicated the presence of varying numbers of differently sized intracellular vacuoles during both normoxic and hyperoxic conditions. CONCLUSIONS: We conclude that the air-liquid interface culture of Calu-3 may be helpful in understanding mechanisms of lung injuries caused in clinical practice, and PFB protects against hyperoxia-induced airway epithelial cell injury by promoting cellular integrity as well as cytologic modifications. PFB-liquid interface culture of Calu-3 may be a useful in vitro model for studying the cytoprotective role of liquid ventilation.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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