Age-Related Impairment of Visual Recognition Memory Correlates with Impaired Synaptic Distribution of GluA2 and Protein Kinase M in the Dentate Gyrus.

Abstract Age-related functional alterations in the perforant path projection from the entorhinal cortex to the dentate gyrus (DG) of the hippocampus play a major role in age-related memory impairments, but little is known about the molecular mechanisms responsible for these changes. In a recent stud...

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Publicado en:Rejuvenation Research Vol. 15; no. 5; pp. 530 - 534
Autor principal: Aicardi G
Formato: Journal Article
Publicado: Mary Ann Liebert, Inc. 2012 Oct
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2012 Oct
      vid: 15
      iid: 5
      pid: 1365
      pub: Mary Ann Liebert, Inc.
      place: New Rochelle, New York
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        108073855
        2011726114
        10.1089/rej.2012.1375
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        atl: Age-Related Impairment of Visual Recognition Memory Correlates with Impaired Synaptic Distribution of GluA2 and Protein Kinase M in the Dentate Gyrus.
      aug:
        au: Aicardi G
        affil: Department of Human and General Physiology, and Interdepartmental Centre ''Luigi Galvani'' for the Study of Biophysics, Bioinformatics and Biocomplexity. University of Bologna , Bologna, Italy .
      sug:
        subj:
          Aging Physiology
          Hippocampus Metabolism
          Memory
          Nervous System Metabolism
          Receptors, Cell Surface Metabolism
          Recognition (Psychology)
          Visual Perception
          Aging Psychosocial Factors
      ab: Abstract Age-related functional alterations in the perforant path projection from the entorhinal cortex to the dentate gyrus (DG) of the hippocampus play a major role in age-related memory impairments, but little is known about the molecular mechanisms responsible for these changes. In a recent study, young and aged monkeys were tested on the visual recognition memory test 'delayed nonmatching-to-sample'; then, electron microscopic immunocytochemistry was performed in the hippocampal DG to determine the subcellular localization of the GluA2 subunit of the glutamate [alpha]-amino-3-hydroxy-5-methyl-4- isoxazole-propionic acid receptor (AMPAR) and protein kinase M (PKM), which promotes memory storage by regulating GluA2-containing AMPAR trafficking. The results obtained suggest that age-related deficits in visual recognition memory are coupled with impairment in PKM-dependent maintenance of GluA2 at the synapse. Together with previous evidences of the critical role of PKM in memory consolidation, these data render this enzyme an attractive potential therapeutic target for treating age-related memory decline, and support the view that the pharmacological manipulation of AMPAR trafficking in the synapses may provide new insights in the search of memory enhancers for aged individuals, including those affected by Alzheimer disease.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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