Quantitative Analysis Linking Inner Hair Cell Voltage Changes and Postsynaptic Conductance Change: A Modelling Study.

This paper presents a computational model which estimates the postsynaptic conductance change of mammalian Type I afferent peripheral process when airborne acoustic waves impact on the tympanic membrane. A model of the human auditory periphery is used to estimate the inner hair cell potential change...

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Publicado en:BioMed Research International Vol. 2015; pp. 1 - 22
Autores principales: Prokopiou, Andreas N., Drakakis, Emm. M.
Formato: equations & formulas pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 1/5/2015
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 1/5/2015
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2015/626971
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        atl: Quantitative Analysis Linking Inner Hair Cell Voltage Changes and Postsynaptic Conductance Change: A Modelling Study.
      aug:
        au:
          Prokopiou, Andreas N.
          Drakakis, Emm. M.
        affil: Department of Bioengineering, Imperial College London, Exhibition Road, London SW7 2AZ, UK
      sug:
        subj:
          Hearing Physiology
          Ear, Inner
          Hair Cells
          Electricity
          Human
          United Kingdom
          Academic Medical Centers
          Quantitative Studies
      ab: This paper presents a computational model which estimates the postsynaptic conductance change of mammalian Type I afferent peripheral process when airborne acoustic waves impact on the tympanic membrane. A model of the human auditory periphery is used to estimate the inner hair cell potential change in response to airborne sound. A generic and tunable topology of the mammalian synaptic ribbon is generated and the voltage dependence of its substructures is used to calculate discrete and probabilistic neurotransmitter vesicle release. Results suggest an almost linear relationship between increasing sound level (in dB SPL) and the postsynaptic conductance for frequencies considered too high for neurons to phase lock with (i.e., a few kHz). Furthermore coordinated vesicle release is shown for up to 300–400 Hz and a mechanism of phase shifting the subharmonic content of a stimulating signal is suggested. Model outputs suggest that strong onset response and highly synchronised multivesicular release rely on compound fusion of ribbon tethered vesicles.
      pubtype: Academic Journal
      doctype:
        equations & formulas
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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