Regulation of Angiogenic Functions by Angiopoietins through Calcium-Dependent Signaling Pathways.

Angiopoietins are vascular factors essential for blood vessel assembly and correct organization and maturation. This study describes a novel calcium-dependent machinery activated through Angiopoietin-1/2-Tie receptor system in HUVECs monolayer. Both cytokines were found to elicit intracellular calci...

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Publicado en:BioMed Research International Vol. 2015; pp. 1 - 15
Autores principales: Pafumi, Irene, Favia, Annarita, Gambara, Guido, Papacci, Francesca, Ziparo, Elio, Palombi, Fioretta, Filippini, Antonio
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 6/4/2015
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 6/4/2015
      vid: 2015
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2015/965271
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        atl: Regulation of Angiogenic Functions by Angiopoietins through Calcium-Dependent Signaling Pathways.
      aug:
        au:
          Pafumi, Irene
          Favia, Annarita
          Gambara, Guido
          Papacci, Francesca
          Ziparo, Elio
          Palombi, Fioretta
          Filippini, Antonio
        affil: Department of Anatomy, Histology, Forensic Medicine and Orthopaedics, Unit of Histology and Medical Embryology, Sapienza University of Rome, 00161 Rome, Italy
      sug:
        subj:
          Signal Transduction
          Neovascularization, Physiologic
          Proteins Metabolism
          Calcium Metabolism
          Human
          Umbilical Veins
          Cell Culture Techniques
          Phosphorylation
          Cytokines
          Growth Substances
          Funding Source
      ab: Angiopoietins are vascular factors essential for blood vessel assembly and correct organization and maturation. This study describes a novel calcium-dependent machinery activated through Angiopoietin-1/2-Tie receptor system in HUVECs monolayer. Both cytokines were found to elicit intracellular calcium mobilization. Targeting intracellular Ca2+ signaling, antagonizing IP3 with 2-APB or cADPR with 8Br-cADPR, was found to modulate in vitro angiogenic responses to Angiopoietins in a specific way. 2-APB and 8Br-cADPR impaired the phosphorylation of AKT and FAK induced by Ang-1 and Ang-2. On the other hand, phosphorylation of ERK1/2 and p38, as well as cell proliferation, was not affected by either inhibitor. The ability of ECs to migrate following Angs stimulation, evaluated by “scratch assay,” was reduced by either 2-APB or 8Br-cADPR following Ang-2 stimulation and only slightly affected by 2-APB in cells stimulated with Ang-1. These results identify a novel calcium-dependent machinery involved in the complex interplay regulating angiogenic processes showing that IP3- and cADPR-induced Ca2+ release specifically regulates distinct Angs-mediated angiogenic steps.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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