Antral gastric permeability to antigens in mice is altered by infection with Helicobacter felis.

Objective: Gastric inflammation is observed not only during Helicobacter pylori infection but also after eradication of the bacterium. The hypothesis that an altered gastric permeability could be involved was tested using a model of mice infected with Helicobacter felis.Design: The antral and corpus...

Descripción completa

Detalles Bibliográficos
Publicado en:European Journal of Gastroenterology & Hepatology Vol. 11; no. 12; pp. 1371 - 1378
Autores principales: Matysiak-Budnik, Tamara, Hashimoto, Kei, heyman, Martine, de mascarel, Antoine, Desjeux, Jehan-Francois, Mégraud, Francis, Matysiak-Budnik, T, Hashimoto, K, Heyman, M, de Mascarel, A, Desjeux, J F, Mégraud, F
Formato: research Journal Article
Publicado: Lippincott Williams & Wilkins Dec1999
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=110639809&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 110639809
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        0954691X
        8CE
      jtl: European Journal of Gastroenterology & Hepatology
      issn: 0954691X
      maglogo: N
    pubinfo:
      dt: Dec1999
      vid: 11
      iid: 12
      pid: 5086
      pub: Lippincott Williams & Wilkins
      place: Baltimore, Maryland
    artinfo:
      ui:
        110639809
        110639809
        NLM10654797
        110639809
        10.1097/00042737-199912000-00006
        NLM10654797
        110639809
      ppf: 1371
      ppct: 7
      formats:
      tig:
        atl: Antral gastric permeability to antigens in mice is altered by infection with Helicobacter felis.
      aug:
        au:
          Matysiak-Budnik, Tamara
          Hashimoto, Kei
          heyman, Martine
          de mascarel, Antoine
          Desjeux, Jehan-Francois
          Mégraud, Francis
          Matysiak-Budnik, T
          Hashimoto, K
          Heyman, M
          de Mascarel, A
          Desjeux, J F
          Mégraud, F
        affil: INSERM E9925, Faculté Necker, 156 rue de vaugirard, 75730 Paris, France
      sug:
        subj:
          Gastric Mucosa Metabolism
          Helicobacter Infections Metabolism
          Antigens Metabolism
          Biological Transport
          Helicobacter Infections Microbiology
          Animal Studies
          Permeability
          Helicobacter
          Gastritis Etiology
          Pyloric Antrum Metabolism
          Mice
          Pyloric Antrum Microbiology
          Urease Metabolism
          Gastric Mucosa Microbiology
          Gastric Mucosa
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
      ab: Objective: Gastric inflammation is observed not only during Helicobacter pylori infection but also after eradication of the bacterium. The hypothesis that an altered gastric permeability could be involved was tested using a model of mice infected with Helicobacter felis.Design: The antral and corpus gastric permeability during infection and after eradication of bacteria was studied.Methods: Gastric fragments from the antrum and corpus of healthy mice, mice infected with H. felis, or mice after bacterial eradication, were mounted in Ussing chambers, and fluxes of sodium (JNa), mannitol (JMan) and horseradish peroxidase (HRP) under intact (JHRPi) and degraded (JD) form were measured.Results: In healthy mice, JNa, JMan, JHRPi and JD, respectively, were greater in the antrum (6.5 +/- 0.5 microEq/h.cm2; 0.137 +/- 0.016 micromol/h.cm2; 30.4 +/- 7.4 ng/h.cm2 and 852 +/- 173 ng/h.cm2) than in the corpus (5.0 +/- 0.3 microEq/h.cm2; 0.085 micro 0.013 micromol/h.cm2; 9.5 +/- 2.8 ng/h.cm2 and 434 +/- 139 ng/h.cm2). In H. felis-infected mice, HRP fluxes in the antrum were increased (JHRPi = 182 +/- 86, JD = 948 +/- 94 ng/h.cm2) as compared to controls (JHRPi = 10.3 +/- 2.6, JD = 458 +/- 98 ng/h.cm2). Bacterial eradication led to the reduction of intact (JHRPi = 53 +/- 26 ng/h.cm) but not of degraded (JD = 844 +/- 213 ng/h.cm) HRP fluxes. After eradication, degraded HRP fluxes returned to normal in mice without inflammation (JD = 558 +/- 36 ng/h.cm2) but not in those with persistent inflammation (JD = 987 +/- 310 ng/h.cm2).Conclusions: The results suggest that during H. felis infection, bacterial colonization and inflammation lead to an increased gastric permeability along the direct and degradative pathways, respectively. Such an increased antigenic load could contribute to the perpetuation of gastric inflammation after bacterial eradication, and possibly to food protein sensitization.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N