The Plasminogen System and Transforming Growth Factor-β in Subjects With Obstructive Sleep Apnea Syndrome: Effects of CPAP Treatment.

BACKGROUND: Obstructive sleep apnea syndrome (OSAS) has emerged as a risk factor for cardiovascular disease. A prothrombotic state may affect coagulation and participate in the atherosclerotic process in subjects with OSAS. These alterations in coagulation seem to involve the plasminogen activation...

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Publicado en:Respiratory Care Vol. 60; no. 11; pp. 1643 - 1652
Autores principales: Steffanina, Alessia, Proietti, Lidia, Antonaglia, Caterina, Palange, Paolo, Angelici, Elena, Canipari, Rita
Formato: research tables/charts Journal Article
Publicado: Mary Ann Liebert, Inc. Nov2015
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Nov2015
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      pub: Mary Ann Liebert, Inc.
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        atl: The Plasminogen System and Transforming Growth Factor-β in Subjects With Obstructive Sleep Apnea Syndrome: Effects of CPAP Treatment.
      aug:
        au:
          Steffanina, Alessia
          Proietti, Lidia
          Antonaglia, Caterina
          Palange, Paolo
          Angelici, Elena
          Canipari, Rita
        affil: Laboratory of Respiratory Pathophysiology, Department of Public Health and Infectious Diseases, Rome, Italy
      sug:
        subj:
          Sleep Apnea, Obstructive
          Peptides Blood
          Plasminogen Activators Blood
          Continuous Positive Airway Pressure
          Human
          Male
          Female
          Post Hoc Analysis
          T-Tests
          Spearman's Rank Correlation Coefficient
          P-Value
          Data Analysis Software
          Growth Substances
          Treatment Outcomes
          Fibrinolysis
          Oxygen Saturation
          Adult
          Middle Age
          Body Mass Index
          Clinical Assessment Tools
          Cardiovascular Risk Factors
          Adult: 19-44 years
          Middle Aged: 45-64 years
          Male
          Female
      ab: BACKGROUND: Obstructive sleep apnea syndrome (OSAS) has emerged as a risk factor for cardiovascular disease. A prothrombotic state may affect coagulation and participate in the atherosclerotic process in subjects with OSAS. These alterations in coagulation seem to involve the plasminogen activation system. We evaluated the imbalances of the plasminogen activation system related to OSAS, and we assessed the effects of CPAP on the plasminogen activation system. METHODS: Thirty-nine subjects were submitted to a home-based cardiorespiratory sleep study, and 14 healthy subjects (apnea-hypopnea index < 5) were used as controls. Serum levels of urokinase-type plasminogen activator (uPA), tissue-type plasminogen activator (tPA), plasminogen activator inhibitor-1 (PAI-1), and active transforming growth factor-β (TGF-β) were measured. These molecules were reassessed in only 17 of the subjects after 1 month of CPAP. RESULTS: PAI-1 and tPA were significantly higher in the subjects with OSAS compared with the controls, whereas TGF-β and uPA levels were lower. PAI-1 showed a significant positive correlation with the apnea-hypopnea index, percentage of time spent at 02 saturation < 90%, and oxygen desaturation index, whereas TGF-β was inversely related to all 3 of these parameters. After the CPAP therapy, PAI-1 significantly decreased, whereas TGF-β showed a significant increase, although the values did not reach those of the controls. uPA and tPA did not show significant differences after the treatment. CONCLUSIONS: Our results suggest an imbalance of fibrinolysis related to OSAS and an improvement of the prothrombotic state after the CPAP treatment.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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