Hepatocyte Transplantation Attenuates the Course of Acute Liver Failure Induced by Thioacetamide in Lewis Rats.

Acute liver failure (ALF) is a clinical syndrome resulting from widespread damage of hepatocytes, with extremely high mortality rate. Urgent orthotopic liver transplantation was shown to be the most effective therapy for ALF but this treatment option is limited by scarcity of donor organs. Therefore...

Descripción completa

Detalles Bibliográficos
Publicado en:Physiological Research Vol. 64; no. 5; pp. 689 - 701
Autores principales: KOBLIHOVÁ, E., LUKŠAN, O., MRÁZOVÁ, I., RYSKA, M., ČERVENKA, L.
Formato: Journal Article
Publicado: Institute of Physiology, Academy of Sciences of the Czech Republic 2015
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=111720371&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 111720371
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        08628408
        1CVW
      jtl: Physiological Research
      issn: 08628408
      maglogo: N
    pubinfo:
      dt: 2015
      vid: 64
      iid: 5
      pid: 24634
      pub: Institute of Physiology, Academy of Sciences of the Czech Republic
    artinfo:
      ui:
        111720371
        10.33549/physiolres.932914
        111720371
      ppf: 689
      ppct: 12
      formats:
        fmt:
          @attributes:
            type: P
      tig:
        atl: Hepatocyte Transplantation Attenuates the Course of Acute Liver Failure Induced by Thioacetamide in Lewis Rats.
      aug:
        au:
          KOBLIHOVÁ, E.
          LUKŠAN, O.
          MRÁZOVÁ, I.
          RYSKA, M.
          ČERVENKA, L.
        affil: Department of Surgery, Second Faculty of Medicine, Charles University and Central Military Hospital, Prague, Czech Republic
      sug:
      ab: Acute liver failure (ALF) is a clinical syndrome resulting from widespread damage of hepatocytes, with extremely high mortality rate. Urgent orthotopic liver transplantation was shown to be the most effective therapy for ALF but this treatment option is limited by scarcity of donor organs. Therefore, hepatocyte transplantation (Tx) has emerged as a new therapeutical measure for ALF, however, the first clinical applications proved unsatisfactory. Apparently, extensive preclinical studies are needed. Our aim was to examine if hepatocytes isolated from transgenic "firefly luciferase" Lewis rats into the recipient liver would attenuate the course of thioacetamide (TAA)-induced ALF in Lewis rats. Untreated Lewis rats after TAA administration showed a profound decrease in survival rate; no animal survived 54 h. The rats showed marked increases in plasma alanine aminotransferase (ALT) and aspartate aminotransferase (AST) activities, in plasma level of bilirubin and ammonia (NH3), and in a significant decrease in plasma albumin. Hepatocyte Tx attenuated the course of TAA-induced ALF Lewis rats which was reflected by improved survival rate and reduced degree of liver injury showing as lowering of elevated plasma ALT, AST, NH3 and bilirubin levels and increasing plasma albumin. In addition, bioluminescence imaging analyses have shown that in the TAA-damaged livers the transplanted hepatocyte were fully viable throughout the experiment. In conclusion, the results show that hepatocyte Tx into the liver can attenuate the course of TAA-induced ALF in Lewis rats. This information should be considered in attempts to develop new therapeutic approaches to the treatment of ALF.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N