Regulation of SCN3B/scn3b by Interleukin 2 (IL-2): IL-2 modulates SCN3B/scn3b transcript expression and increases sodium current in myocardial cells.

Background: In the initiation and maintenance of arrhythmia, inflammatory processes play an important role. IL-2 is a pro-inflammatory factor which is associated with the morbidity of arrhythmias, however, how IL-2 affects the cardiac electrophysiology is still unknown.Methods: In the present study,...

Descripción completa

Detalles Bibliográficos
Publicado en:BMC Cardiovascular Disorders Vol. 16; pp. 1 - 8
Autores principales: Yuanyuan Zhao, Qiaobing Sun, Zhipeng Zeng, Qianqian Li, Shiyuan Zhou, Mengchen Zhou, Yumei Xue, Xiang Cheng, Yunlong Xia, Qing Wang, Xin Tu, Zhao, Yuanyuan, Sun, Qiaobing, Zeng, Zhipeng, Li, Qianqian, Zhou, Shiyuan, Zhou, Mengchen, Xue, Yumei, Cheng, Xiang, Xia, Yunlong
Formato: pictorial research tables/charts Journal Article
Publicado: BioMed Central 1/4/2016
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=112086398&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 112086398
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        14712261
        1CHA
      jtl: BMC Cardiovascular Disorders
      issn: 14712261
      maglogo: N
    pubinfo:
      dt: 1/4/2016
      vid: 16
      pid: 24147
      pub: BioMed Central
    artinfo:
      ui:
        112086398
        112086398
        NLM26728597
        112086398
        10.1186/s12872-015-0179-x
        NLM26728597
        PMC4700781
        112086398
      ppf: 1
      ppct: 7
      formats:
      tig:
        atl: Regulation of SCN3B/scn3b by Interleukin 2 (IL-2): IL-2 modulates SCN3B/scn3b transcript expression and increases sodium current in myocardial cells.
      aug:
        au:
          Yuanyuan Zhao
          Qiaobing Sun
          Zhipeng Zeng
          Qianqian Li
          Shiyuan Zhou
          Mengchen Zhou
          Yumei Xue
          Xiang Cheng
          Yunlong Xia
          Qing Wang
          Xin Tu
          Zhao, Yuanyuan
          Sun, Qiaobing
          Zeng, Zhipeng
          Li, Qianqian
          Zhou, Shiyuan
          Zhou, Mengchen
          Xue, Yumei
          Cheng, Xiang
          Xia, Yunlong
        affil: Key Laboratory of Molecular Biophysics of Ministry of Education, College of Life Science and Technology, Center for Human Genome Research, Cardio-X Institute, Huazhong University of Science and Technology, Wuhan 430074, China
      sug:
        subj:
          RNA Pharmacodynamics
          Interleukin 2 Drug Effects
          Genes
          Calcium Drug Effects
          Cells Metabolism
          Sodium Drug Effects
          Sodium Metabolism
          Genes Drug Effects
          Epithelial Cells Drug Effects
          Potassium Metabolism
          Cells
          Reverse Transcriptase Polymerase Chain Reaction
          Potassium Drug Effects
          Calcium Metabolism
          Sodium Pharmacodynamics
          RNA
          Cells Drug Effects
          Epithelial Cells
          Potassium Pharmacodynamics
          Membrane Potentials
          RNA Drug Effects
          Cytological Techniques
          Epithelial Cells Metabolism
          Interleukin 2 Metabolism
          Calcium Pharmacodynamics
          Interleukin 2 Pharmacodynamics
          Membrane Potentials Drug Effects
          Sodium
          Potassium
          RNA Metabolism
          Calcium
          Interleukin 2
      ab: Background: In the initiation and maintenance of arrhythmia, inflammatory processes play an important role. IL-2 is a pro-inflammatory factor which is associated with the morbidity of arrhythmias, however, how IL-2 affects the cardiac electrophysiology is still unknown.Methods: In the present study, we observed the effect of IL-2 by qRT-PCR on the transcription of ion channel genes including SCN2A, SCN3A, SCN4A, SCN5A, SCN9A, SCN10A, SCN1B, SCN2B, SCN3B, KCNN1, KCNJ5, KCNE1, KCNE2, KCNE3, KCND3, KCNQ1, KCNA5, KCNH2 and CACNA1C. Western blot assays and electrophysiological studies were performed to demonstrate the effect of IL-2 on the translation of SCN3B/scn3b and sodium currents.Results: The results showed that transcriptional level of SCN3B was up-regulated significantly in Hela cells (3.28-fold, p = 0.022 compared with the control group). Consistent results were verified in HL-1 cells (3.73-fold, p = 0.012 compared with the control group). The result of electrophysiological studies showed that sodium current density increased significantly in cells which treated by IL-2 and the effect of IL-2 on sodium currents was independent of SCN3B (1.4 folds, p = 0.023). Western blot analysis showed IL-2 lead to the significantly increasing of p53 and scn3b (2.1 folds, p = 0.021 for p53; 3.1 folds, p = 0.023 for scn3b) in HL-1 cells. Consistent results were showed in HEK293 cells using qRT-PCR analysis (1.43 folds for P53, p = 0.022; 1.57 folds for SCN3B, p = 0.05).Conclusions: The present study suggested that IL-2, may play role in the arrhythmia by regulating the expression of SCN3B and sodium current density.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N