Low-Level Vagus Nerve Stimulation Attenuates Myocardial Ischemic Reperfusion Injury by Antioxidative Stress and Antiapoptosis Reactions in Canines.

Vagus Nerve Stimulation and Myocardial Ischemia-Reperfusion Injury Background Low-level vagus nerve stimulation (LL-VNS) has been demonstrated to protect myocardium against acute ischemia/reperfusion (I/R) injury. However, the underlying mechanism of this protective effect remains unknown. Objective...

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Publicado en:Journal of Cardiovascular Electrophysiology Vol. 27; no. 2; pp. 224 - 232
Autores principales: CHEN, MINGXIAN, ZHOU, XIAOYA, YU, LILEI, LIU, QIMING, SHENG, XIA, WANG, ZHUO, WANG, SONGYUN, JIANG, HONG, ZHOU, SHENGHUA
Formato: pictorial research tables/charts tracings Journal Article
Publicado: Wiley-Blackwell Feb2016
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Feb2016
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        atl: Low-Level Vagus Nerve Stimulation Attenuates Myocardial Ischemic Reperfusion Injury by Antioxidative Stress and Antiapoptosis Reactions in Canines.
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          CHEN, MINGXIAN
          ZHOU, XIAOYA
          YU, LILEI
          LIU, QIMING
          SHENG, XIA
          WANG, ZHUO
          WANG, SONGYUN
          JIANG, HONG
          ZHOU, SHENGHUA
        affil: Department of Cardiology, The Second Xiangya Hospital of Central South University, Changsha Hunan, China
      sug:
        subj:
          Vagal Stimulation Utilization
          Reperfusion Injury Prevention and Control
          Myocardial Ischemia Prevention and Control
          Animal Studies
          Dogs
          Random Assignment
          Dyes Diagnostic Use
          Funding Source
          Oxidative Stress Physiology
          Heart Rate Variability
          Spectral Analysis
          Descriptive Statistics
          Data Analysis Software
          P-Value
          Confidence Intervals
          Odds Ratio
          Algorithms
          Severity of Injury Evaluation
          Myocardial Infarction
          Neutrophils Immunology
          Study Design
          T-Tests
          Fisher's Exact Test
          One-Way Analysis of Variance
          Staining and Labeling
          Apoptosis Physiology
          Gene Expression
          Blotting, Western Utilization
          Oncogenes Physiology
          Proteins Physiology
          Histocytochemistry Methods
      ab: Vagus Nerve Stimulation and Myocardial Ischemia-Reperfusion Injury Background Low-level vagus nerve stimulation (LL-VNS) has been demonstrated to protect myocardium against acute ischemia/reperfusion (I/R) injury. However, the underlying mechanism of this protective effect remains unknown. Objective This study aimed to test the hypothesis that LL-VNS exerts cardioprotective effect on acute I/R injury in canines via antioxidative stress and antiapoptosis reactions. Method Thirty anesthetized mongrel dogs were randomly divided into three groups: I/R group (N = 12, the left anterior descending coronary artery was occluded for 1 hour following by 1 hour reperfusion), LL-VNS group (N = 9, I/R plus LL-VNS), and sham group (N = 9, sham surgery without LL-VNS). The voltage threshold was set at 80% of the voltage required to slow the sinus rate. Infarct size was assessed with Evans Blue and triphenyltetrazolium chloride. Activity assays, TUNEL staining, and western blotting were performed to determine markers of oxidative stress and apoptosis. Results LL-VNS significantly decreased the incidence of ventricular arrhythmias, increased vagal tone, as confirmed by heart rate viability, and reduced infarct size compared with the I/R group. This improvement was associated with a reduction in myocardial neutrophil infiltration, the inhibition of oxidative stress, and the suppression in cardiomyocyte apoptosis. In contrast, the lack of LL-VNS in the I/R group induced the opposite effect compared with the sham group. Conclusion LL-VNS exerts protective effects on myocardial I/R injury. Its potential mechanisms involve the suppression of oxidative stress and cellular apoptosis.
      pubtype: Academic Journal
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        pictorial
        research
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      ougenre: Article
    language: English
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