Study of Biofilm Formation in C57BL/6J Mice by Clinical Isolates of Helicobacter pylori.

Background/Aim: Despite the significant number of studies on H. pylori pathogenesis, not much data has been published concerning its ability to form biofilm in the host stomach. This study aims to evaluate the potential of clinical isolates of H. pylori to form biofilm in C57BL/ 6J mice model. Mater...

Descripción completa

Detalles Bibliográficos
Publicado en:Saudi Journal of Gastroenterology Vol. 31; no. 2; pp. 161 - 169
Autores principales: Attaran, Bahareh, Falsafi, Tahereh, Moghaddam, Ali N.
Formato: research tables/charts Journal Article
Publicado: Wolters Kluwer India Pvt Ltd Apr-Jun2016
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=113935673&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 113935673
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        13193767
        322P
      jtl: Saudi Journal of Gastroenterology
      issn: 13193767
      maglogo: N
    pubinfo:
      dt: Apr-Jun2016
      vid: 31
      iid: 2
      pid: 16919
      pub: Wolters Kluwer India Pvt Ltd
    artinfo:
      ui:
        113935673
        113935673
        113935673
        10.4103/1319-3767.178529
        113935673
      ppf: 161
      ppct: 8
      formats:
      tig:
        atl: Study of Biofilm Formation in C57BL/6J Mice by Clinical Isolates of Helicobacter pylori.
      aug:
        au:
          Attaran, Bahareh
          Falsafi, Tahereh
          Moghaddam, Ali N.
        affil: Department of Microbiology, School of Biology, Alzahra University
      sug:
        subj:
          Biofilms
          Helicobacter Infections Pathology
          Stomach Microbiology
          Mice
          Animal Studies
          Staining and Labeling
          Gastrostomy Tubes
          Bacterial Colonization
          Urease
          Enzyme-Linked Immunosorbent Assay
          Bacterial Infections Pathology
          Fluorescent Antibody Technique
          Microscopy, Electron, Scanning
          In Vitro Studies
          Immunoglobulins
          Epithelium Pathology
          Gastrointestinal System Pathology
          Gastric Mucosa Microbiology
      ab: Background/Aim: Despite the significant number of studies on H. pylori pathogenesis, not much data has been published concerning its ability to form biofilm in the host stomach. This study aims to evaluate the potential of clinical isolates of H. pylori to form biofilm in C57BL/ 6J mice model. Materials and Methods: Two strains of H. pylori were selected from a collection of clinical isolates; one (19B), an efficient biofilm producer and the other (4B), with weak biofilm-forming ability. Mice infected through gastric gavages were examined after one and two weeks. Colonization was determined by CFU and urease activity; the anti-H. pylori IgA was measured by ELISA, and chronic infections were evaluated by histopathology. Bacterial communities within mucosal sections were studied by immunofluorescence and scanning electron microscopy (SEM). Results: Successful infection was obtained by both test strains. Strain 19B with higher ability to form biofilm in vitro also showed a higher colonization rate in the mice stomach one week after infection. Difference (P < 0.05) in IgA titers was observed between the infected mice and the controls as well as between 19B and 4B infected mice, two weeks after the last challenge. Immunofluorescence and SEM results showed tightly colonizing H. pylori in stomach mucosal sections and in squamous and glandular epithelium. Conclusion: H. pylori is able to form biofilm in the mouse stomach and induce IgA production, reflecting the same potential as in humans. Firm attachment of coccoid form bacteria to host cells suggests the importance of this state in biofilm formation by H. pylori. Occurrence of biofilm in squamous and glandular epithelium of the mouse stomach proposes that H. pylori can colonize all parts of the upper gastrointestinal tract.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N