PASS Syndrome: An IL-1-Driven Autoinflammatory Disease.

PASS syndrome is a rare inflammatory disease characterized by a chronic-relapsing course of pyoderma gangrenosum, acne vulgaris, hidradenitis suppurativa and ankylosing spondylitis. Here, we describe a case of a patient with spontaneously recurrent purulent skin lesions along with seronegative spond...

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Publicado en:Dermatology (10188665) Vol. 232; no. 2; pp. 254 - 259
Autores principales: Leuenberger, Mathieu, Berner, Jeanne, Di Lucca, Julie, Fischer, Lara, Kaparos, Nikolaos, Conrad, Curdin, Hohl, Daniel, So, alexander, Gilliet, Michel
Formato: case study Journal Article
Publicado: Karger AG Apr2016
Acceso en línea:Ver este registro en EBSCOhost
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      pub: Karger AG
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        atl: PASS Syndrome: An IL-1-Driven Autoinflammatory Disease.
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        au:
          Leuenberger, Mathieu
          Berner, Jeanne
          Di Lucca, Julie
          Fischer, Lara
          Kaparos, Nikolaos
          Conrad, Curdin
          Hohl, Daniel
          So, alexander
          Gilliet, Michel
        affil: Dermatology
      sug:
        subj:
          Autoimmune Diseases Drug Therapy
          Antirheumatic Agents Therapeutic Use
          Acne Vulgaris Diagnosis
          Proteins Therapeutic Use
          Spondylitis, Ankylosing Diagnosis
          Hidradenitis Suppurativa Diagnosis
          Pyoderma Gangrenosum Diagnosis
          Autoimmune Diseases Diagnosis
          Male
          Autoimmune Diseases Blood
          Syndrome
          Interleukin 1 Blood
          Adult
          Psychological Tests
          Adult: 19-44 years
          Male
      ab: PASS syndrome is a rare inflammatory disease characterized by a chronic-relapsing course of pyoderma gangrenosum, acne vulgaris, hidradenitis suppurativa and ankylosing spondylitis. Here, we describe a case of a patient with spontaneously recurrent purulent skin lesions along with seronegative spondylarthritis consistent with the PASS syndrome. During his disease exacerbation, the patient displayed episodes of fever along with elevated serum levels of interleukin (IL)-1β. Skin lesions were characterized by sterile neutrophilic infiltrates and showed a rapid response to the IL-1 receptor antagonist anakinra (Kineret®) consistent with the autoinflammatory nature of this disease. However, unlike other autoinflammatory diseases such as PAPA and PAPASH, we did not find mutations in the gene PSTPIP1, raising the possibility that other specific mutations in the IL-1 pathway may be involved.
      pubtype: Academic Journal
      doctype:
        case study
        Journal Article
      ougenre: Article
    language: English
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