Unclassified renal cell carcinoma with tubulopapillary architecture, clear cell phenotype, and chromosome 8 monosomy: a new kid on the block.

Accurate subtyping of renal cell carcinomas (RCCs) has become clinically important for therapy and prognostication. RCC subtypes are defined by distinct morphologic and immunohistochemical profiles, and in some instances recurrent cytogenetic and molecular properties. However, some tumors exhibit ov...

Descripción completa

Detalles Bibliográficos
Publicado en:Virchows Archiv: European Journal of Pathology Vol. 469; no. 1; pp. 81 - 92
Autores principales: Lan, Thanh, Keller-Ramey, Jennifer, Fitzpatrick, Carrie, Kadri, Sabah, Taxy, Jerome, Segal, Jeremy, Furtado, Larissa, Antic, Tatjana, Lan, Thanh T H, Taxy, Jerome B, Segal, Jeremy P, Furtado, Larissa V
Formato: Journal Article
Publicado: Springer Nature Jul2016
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=116415193&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 116415193
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        09456317
        O1Z
      jtl: Virchows Archiv: European Journal of Pathology
      issn: 09456317
      maglogo: N
    pubinfo:
      dt: Jul2016
      vid: 469
      iid: 1
      pid: 237
      pub: Springer Nature
      place: New York, New York
    artinfo:
      ui:
        116415193
        116415193
        NLM27173781
        10.1007/s00428-016-1952-7
        NLM27173781
        116415193
      ppf: 81
      ppct: 11
      formats:
        fmt:
          @attributes:
            type: P
      tig:
        atl: Unclassified renal cell carcinoma with tubulopapillary architecture, clear cell phenotype, and chromosome 8 monosomy: a new kid on the block.
      aug:
        au:
          Lan, Thanh
          Keller-Ramey, Jennifer
          Fitzpatrick, Carrie
          Kadri, Sabah
          Taxy, Jerome
          Segal, Jeremy
          Furtado, Larissa
          Antic, Tatjana
          Lan, Thanh T H
          Taxy, Jerome B
          Segal, Jeremy P
          Furtado, Larissa V
        affil: Department of Pathology, The University of Chicago, 5841 S. Maryland Avenue Chicago 60637 USA
      sug:
        subj:
          Chromosome Disorders Pathology
          Chromosomes
          Carcinoma, Renal Cell Pathology
          Carcinoma, Renal Cell
          Mutation
          Aged
          Carcinoma, Renal Cell Metabolism
          Chromosome Disorders
          In Situ Hybridization, Fluorescence Methods
          Middle Age
          Female
          Phenotype
          Male
          Aged, 80 and Over
          Adult
          Aged: 65+ years
          Middle Aged: 45-64 years
          Aged, 80 & over
          Adult: 19-44 years
          Female
          Male
      ab: Accurate subtyping of renal cell carcinomas (RCCs) has become clinically important for therapy and prognostication. RCC subtypes are defined by distinct morphologic and immunohistochemical profiles, and in some instances recurrent cytogenetic and molecular properties. However, some tumors exhibit overlapping morphologic and immunophenotypic features, frequent enough to pose diagnostic dilemmas. This report concerns six histologically unusual RCCs that showed tubulopapillary architecture, clear cell phenotype, and non-diagnostic immunohistochemical profiles. Further investigation of these tumors utilized a single nucleotide polymorphism (SNP) microarray platform (OncoScan®, Affymetrix) that employed molecular inversion probe (MIP) technology to investigate genome-wide chromosomal copy number changes and loss of heterozygosity in formalin-fixed paraffin-embedded sections. The six tumors were assayed in parallel with and in comparison to RCC with typical morphologic or immunohistochemical features for a specific subtype (clear cell, clear cell papillary, and microphthalmia transcription factor (MiT) family translocation RCC). Three of the unusual RCCs showed a molecular signature of clear cell RCC and one of papillary RCC. The remaining two showed monosomy of chromosome 8. Those two cases were tested via next-generation sequencing, and no pathogenic variants were detected, including those in the genes VHL, PBRM1, SETD2, KDM5C, or BAP1. The addition of molecular investigations such as reported here as applied to histologically and immunohistochemically unusual RCC may help to define additional subtypes and contribute to the development of targeted therapy for renal cancer.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N