Role of Ga somatostatin receptor PET/CT in the detection of endogenous hyperinsulinaemic focus: an explorative study.

Purpose: To explore the role of Ga-DOTATATE/DOTATOC PET/CT (SR PET/CT) in patients with suspicion of or histopathologically proven pancreatogenic hyperinsulinaemic hypoglycaemia. Methods: We included 13 patients with histopathologically proven or a high clinical suspicion of pancreatogenic hyperinsu...

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Publicado en:European Journal of Nuclear Medicine & Molecular Imaging Vol. 43; no. 9; pp. 1593 - 1601
Autores principales: Prasad, Vikas, Sainz-Esteban, Aurora, Arsenic, Ruza, Plöckinger, Ursula, Denecke, Timm, Pape, Ulrich-Frank, Pascher, Andreas, Kühnen, Peter, Pavel, Marianne, Blankenstein, Oliver
Formato: Journal Article
Publicado: Springer Nature Aug2016
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Aug2016
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00259-016-3331-7
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        atl: Role of Ga somatostatin receptor PET/CT in the detection of endogenous hyperinsulinaemic focus: an explorative study.
      aug:
        au:
          Prasad, Vikas
          Sainz-Esteban, Aurora
          Arsenic, Ruza
          Plöckinger, Ursula
          Denecke, Timm
          Pape, Ulrich-Frank
          Pascher, Andreas
          Kühnen, Peter
          Pavel, Marianne
          Blankenstein, Oliver
        affil: Department of Nuclear Medicine, Charité Universitätsmedizin Berlin, Berlin Germany
      sug:
      ab: Purpose: To explore the role of Ga-DOTATATE/DOTATOC PET/CT (SR PET/CT) in patients with suspicion of or histopathologically proven pancreatogenic hyperinsulinaemic hypoglycaemia. Methods: We included 13 patients with histopathologically proven or a high clinical suspicion of pancreatogenic hyperinsulinaemia. All the patients underwent a SR PET/CT scan. The results were correlated with histopathological findings. Normalization of blood glucose levels after resection of the pancreatic lesion, as well as a cytological and/or pathological diagnosis of insulinoma, was considered the diagnostic gold standard for insulinoma. The diagnosis of nesidioblastosis was based on exclusion of an insulinoma and conclusive pathological examination of a segment of the pancreas. Malignant insulinoma was defined as the presence of locoregional or distant metastases. Results: Based on histopathology, 13 patients were found to have pancreatic hyperinsulinaemia: two patients had malignant insulinoma, eight had nonmetastasized insulinoma, and three had nesidioblastosis. SR PET was positive in 11 of the 13 patients (84.6 %) with a final diagnosis of endogenous pancreatic hypoglycaemia. Histopathological staining confirmed 16 foci of hyperinsulinism (insulin positivity). SR PET detected 14 of the 16 lesions, resulting in a sensitivity of 87 %. One intrapancreatic spleen was falsely diagnosed as insulinoma focus on SR PET, resulting in positive predictive value of 93.3 %. Immunohistochemical staining of somatostatin receptor (SSR) subtype 2a was available in ten specimens: two nesidioblastosis, and seven benign and one malignant insulinoma. Eight out of the ten specimens (80 %) stained strongly to moderately positive. Seven of the eight SSR2a-positive lesions were picked up on SR PET. Based on the results of SR PET/CT, nine patients achieved complete remission of the hypoglycaemic events during follow-up. Conclusion: This explorative study suggests that SR PET in combination with CT may play a significant role in the detection and management of patients with pancreatogenic hyperinsulinaemic hypoglycaemia. A large proportion of insulinomas express SSR2a, and a larger study is needed to fully assess the diagnostic accuracy of SR PET in patients with insulinoma and nesidioblastosis compared with current localizing studies used in clinical practice.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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