2-Bromoterguride-a potential atypical antipsychotic drug without metabolic effects in rats.

Rationale: Recently, we showed that 2-bromoterguride acted as a dopamine D receptor partial agonist, a serotonin 5-HT and α-adrenergic receptor antagonist, and exhibited antidopaminergic efficacy in amphetamine-induced locomotion (AIL) in rats without inducing catalepsy. Objective: To extend our kno...

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Publicado en:Psychopharmacology Vol. 233; no. 15/16; pp. 3041 - 3051
Autores principales: Franke, Robert, Tarland, Emilia, Fink, Heidrun, Pertz, Heinz, Brosda, Jan
Formato: Journal Article
Publicado: Springer Nature Aug2016
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Aug2016
      vid: 233
      iid: 15/16
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00213-016-4356-0
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        atl: 2-Bromoterguride-a potential atypical antipsychotic drug without metabolic effects in rats.
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        au:
          Franke, Robert
          Tarland, Emilia
          Fink, Heidrun
          Pertz, Heinz
          Brosda, Jan
        affil: Institute of Pharmacology and Toxicology, School of Veterinary Medicine, Freie Universität Berlin, 14195 Berlin Germany
      sug:
      ab: Rationale: Recently, we showed that 2-bromoterguride acted as a dopamine D receptor partial agonist, a serotonin 5-HT and α-adrenergic receptor antagonist, and exhibited antidopaminergic efficacy in amphetamine-induced locomotion (AIL) in rats without inducing catalepsy. Objective: To extend our knowledge on the antipsychotic effects of 2-bromoterguride, we used convergent preclinical animal models and tests; i.e., conditioned avoidance response (CAR), predictive of antipsychotic-like effects; Fos protein expression, a molecular marker for (atypical) antipsychotic activity; wet dog shake behavior, a test for the in vivo effects of drugs acting on central 5-HT receptors; and investigated metabolic changes as a common side effect of atypical antipsychotic drugs (APDs). Results: Acute treatment with 2-bromoterguride (0.1 and 0.3 mg/kg) decreased the CAR at 30, 90, and 270 min post-injection in rats without inducing escape failures at any time. Fos protein expression, as shown by Western blotting, was enhanced by 2-bromoterguride in the nucleus accumbens (NAc), the dorsolateral striatum (dStr), and the medial prefrontal cortex (mPFC). (±)-2,5-Dimethoxy-4-iodoamphetamine (DOI)-induced wet dog shakes in rats were reduced by 2-bromoterguride. Chronic treatment with 2-bromoterguride did not affect metabolic parameters such as body weight development and body fat composition as well as behavioral parameters such as food intake and locomotor activity. Conclusions: Our data suggest that 2-bromoterguride is a promising candidate in the treatment of schizophrenia due to its atypical antipsychotic-like activity and its inability to induce weight gain.
      pubtype: Academic Journal
      doctype: Journal Article
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    language: English
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