Role of miRNAs in Epicardial Adipose Tissue in CAD Patients with T2DM.

Background. Epicardial adipose tissue (EAT) is identified as an atypical fat depot surrounding the heart with a putative role in the involvement of metabolic disorders, including obesity, type-2 diabetes mellitus, and atherosclerosis. We profiled miRNAs in EAT of metabolic patients with coronary art...

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Publicado en:BioMed Research International Vol. 2016; pp. 1 - 8
Autores principales: Liu, Yang, Fu, Wenbo, Lu, Mu, Huai, Shitao, Song, Yaqin, Wei, Yutao
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell 8/14/2016
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 8/14/2016
      vid: 2016
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      pub: Wiley-Blackwell
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        10.1155/2016/1629236
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        atl: Role of miRNAs in Epicardial Adipose Tissue in CAD Patients with T2DM.
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          Liu, Yang
          Fu, Wenbo
          Lu, Mu
          Huai, Shitao
          Song, Yaqin
          Wei, Yutao
        affil: Medicine Department, Shihezi University, Shihezi, Xinjiang 832000, China
      sug:
        subj:
          Diabetes Mellitus, Type 2 Pathology
          Coronary Arteriosclerosis Pathology
          RNA Analysis
          Pericardium
          Adipose Tissue
          RNA Classification
          Human
          Microarray Analysis
          China
          P-Value
          Descriptive Statistics
          Bioinformatics
          Data Analysis Software
          Male
          Female
          Academic Medical Centers
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          Middle Age
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          Wilcoxon Signed Rank Test
          Funding Source
          Aged: 65+ years
          Middle Aged: 45-64 years
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      ab: Background. Epicardial adipose tissue (EAT) is identified as an atypical fat depot surrounding the heart with a putative role in the involvement of metabolic disorders, including obesity, type-2 diabetes mellitus, and atherosclerosis. We profiled miRNAs in EAT of metabolic patients with coronary artery disease (CAD) and type-2 diabetes mellitus (T2DM) versus metabolically healthy patients by microarray. Compared to metabolically healthy patients, we identified forty-two miRNAs that are differentially expressed in patients with CAD and T2DM from Xinjiang, China. Eleven miRNAs were selected as potential novel miRNAs according to P value and fold change. Then the potential novel miRNAs targeted genes were predicted via TargetScan, PicTar, and miRTarbase, and the function of the target genes was predicted via Gene Ontology (GO) analysis while the enriched KEGG pathway analyses of the miRNAs targeted genes were performed by bioinformatics software DAVID. Then protein-protein interaction networks of the targeted gene were conducted by online software STRING. Finally, using microarray, bioinformatics approaches revealed the possible molecular mechanisms pathogenesis of CAD and T2DM. A total of 11 differentially expressed miRNAs were identified and among them, hsa-miR-4687-3p drew specific attention. Bioinformatics analysis revealed that insulin signaling pathway is the central way involved in the progression of metabolic disorders. Conclusions. The current findings support the fact that miRNAs are involved in the pathogenesis of metabolic disorders in EAT of CAD patients with T2DM, and validation of the results of these miRNAs by independent and prospective study is certainly warranted.
      pubtype: Academic Journal
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        research
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      ougenre: Article
    language: English
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