A study of hearing function and histopathologic changes in the cochlea of the type 2 diabetes model Tsumura Suzuki obese diabetes mouse.

Objectives:This study used Tsumura Suzuki Obese Diabetes (TSOD) mice as a spontaneous type 2 diabetes model and Tsumura Suzuki Non-obesity (TSNO) mice as controls to investigate factors involved in the onset of hearing impairment. Method:Body weight, blood glucose levels, and auditory brainstem resp...

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Publicado en:Acta Oto-Laryngologica Vol. 136; no. 11; pp. 1097 - 1107
Autores principales: Tsuda, Junko, Sugahara, Kazuma, Hori, Takeshi, Kanagawa, Eiju, Takaki, Eiichi, Fujimoto, Mitsuaki, Nakai, Akira, Yamashita, Hiroshi
Formato: pictorial research tables/charts Journal Article
Publicado: Taylor & Francis Ltd Nov2016
Acceso en línea:Ver este registro en EBSCOhost
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        10.1080/00016489.2016.1195012
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        atl: A study of hearing function and histopathologic changes in the cochlea of the type 2 diabetes model Tsumura Suzuki obese diabetes mouse.
      aug:
        au:
          Tsuda, Junko
          Sugahara, Kazuma
          Hori, Takeshi
          Kanagawa, Eiju
          Takaki, Eiichi
          Fujimoto, Mitsuaki
          Nakai, Akira
          Yamashita, Hiroshi
        affil: Department of Otolaryngology, Yamaguchi University Graduate School of Medicine, Ube City, Yamaguchi Prefecture, Japan
      sug:
        subj:
          Hearing Disorders
          Cochlea
          Diabetes Mellitus, Type 2
          Obesity
          Animal Studies
          Mice
          Histological Techniques
          In Vivo Studies
          In Vitro Studies
          Staining and Labeling
          Microscopy
          Descriptive Statistics
          Data Analysis Software
          Microarray Analysis
          Mann-Whitney U Test
          Pearson's Correlation Coefficient
          Fisher's Exact Test
          Funding Source
      ab: Objectives:This study used Tsumura Suzuki Obese Diabetes (TSOD) mice as a spontaneous type 2 diabetes model and Tsumura Suzuki Non-obesity (TSNO) mice as controls to investigate factors involved in the onset of hearing impairment. Method:Body weight, blood glucose levels, and auditory brainstem responses (ABRs) were measured. The cochleae were excised and evaluated histopathologically. Results:The TSOD mice showed significant hyperglycemia at 2–7 months and severe obesity at 5–10 months; significantly elevated ABR thresholds at 8–10 months; and the capillary lumens in the cochlea stria vascularis were narrower in the TSOD mice than in the TSNO mice. At 17 months, India ink vascular staining of the TSOD mice’s cochleae revealed decreased capillary density in the stria vascularis. The vascular area of capillaries in the stria vascularis and the vascular area were significantly smaller in TSOD mice. Histopathological analysis showed vessel wall thickening in the modiolus and narrowed capillaries in the stria vascularis, suggesting reduced blood flow to the inner ear. Conclusion:The diabetes mice model used in our study showed early age-associated hearing loss, and histopathology showed findings of vessel wall thickening in the modiolus, narrowing of capillaries in the stria vascularis, and chronically reduced blood flow in the cochlea.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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