Modelling the effect of insulin on the disposal of meal-attributable glucose in type 1 diabetes.

The management of postprandial glucose excursions in type 1 diabetes has a major impact on overall glycaemic control. In this work, we propose and evaluate various mechanistic models to characterize the disposal of meal-attributable glucose. Sixteen young volunteers with type 1 diabetes were subject...

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Published in:Medical & Biological Engineering & Computing Vol. 55; no. 2; pp. 271 - 283
Main Authors: García-García, Fernando, Hovorka, Roman, Wilinska, Malgorzata, Elleri, Daniela, Hernando, M., Wilinska, Malgorzata E, Hernando, M Elena
Format: Journal Article
Published: Springer Nature Feb2017
Online Access:View this record in EBSCOhost
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        atl: Modelling the effect of insulin on the disposal of meal-attributable glucose in type 1 diabetes.
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          García-García, Fernando
          Hovorka, Roman
          Wilinska, Malgorzata
          Elleri, Daniela
          Hernando, M.
          García-García, Fernando
          Wilinska, Malgorzata E
          Hernando, M Elena
        affil: Wellcome Trust-MRC Institute of Metabolic Science , University of Cambridge , Cambridge UK
      sug:
        subj:
          Diabetes Mellitus, Type 1 Metabolism
          Models, Biological
          Insulin Therapeutic Use
          Glucose Metabolism
          Postprandial Period Physiology
          Female
          Young Adult
          Blood Glucose Metabolism
          Insulin Blood
          Models, Theoretical
          Diabetes Mellitus, Type 1 Drug Therapy
          Adolescence
          Reproducibility of Results
          Male
          Impact of Events Scale
          Scales
          Adolescent: 13-18 years
          Female
          Male
      ab: The management of postprandial glucose excursions in type 1 diabetes has a major impact on overall glycaemic control. In this work, we propose and evaluate various mechanistic models to characterize the disposal of meal-attributable glucose. Sixteen young volunteers with type 1 diabetes were subject to a variable-target clamp which replicated glucose profiles observed after a high-glycaemic-load ([Formula: see text]) or a low-glycaemic-load ([Formula: see text]) evening meal. [6,6-[Formula: see text]] and [U-[Formula: see text];1,2,3,4,5,6,6-[Formula: see text]] glucose tracers were infused to, respectively, mimic: (a) the expected post-meal suppression of endogenous glucose production and (b) the appearance of glucose due to a standard meal. Six compartmental models (all a priori identifiable) were proposed to investigate the remote effect of circulating plasma insulin on the disposal of those glucose tracers from the non-accessible compartments, representing e.g. interstitium. An iterative population-based parameter fitting was employed. Models were evaluated attending to physiological plausibility, posterior identifiability of their parameter estimates, accuracy-via weighted fitting residuals-and information criteria (i.e. parsimony). The most plausible model, best representing our experimental data, comprised: (1) a remote effect x of insulin active above a threshold [Formula: see text] = 1.74 (0.81-2.50) [Formula: see text] min[Formula: see text] [median (inter-quartile range)], with parameter [Formula: see text] having a satisfactory support: coefficient of variation CV = 42.33 (31.34-65.34) %, and (2) steady-state conditions at the onset of the experiment ([Formula: see text]) for the compartment representing the remote effect, but not for the masses of the tracer that mimicked endogenous glucose production. Consequently, our mechanistic model suggests non-homogeneous changes in the disposal rates for meal-attributable glucose in relation to plasma insulin. The model can be applied to the in silico simulation of meals for the optimization of postprandial insulin infusion regimes in type 1 diabetes.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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