Molecular Analysis of Central Nervous System Disease Spectrum in Childhood Acute Lymphoblastic Leukemia.

Treatment of the central nervous system (CNS) is an essential therapeutic component in childhood acute lymphoblastic leukemia (ALL). The goal of this study was to identify molecular signatures distinguishing patients with CNS disease from those without the disease in pediatric patients with ALL. We...

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Publicado en:Clinical Medicine Insights: Oncology no. 10; pp. 5 - 16
Autores principales: Hicks, Chindo, Sitthi-Amorn, Jitsuda, Douglas, Jessica, Ramani, Ritika, Miele, Lucio, Vijayakumar, Vani, Karlson, Cynthia, Chipeta, James, Megason, Gail
Formato: pictorial research tables/charts Journal Article
Publicado: Sage Publications Inc. 2016
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2016
      iid: 10
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      pub: Sage Publications Inc.
      place: Thousand Oaks, California
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        10.4137/CMO.s18180
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        atl: Molecular Analysis of Central Nervous System Disease Spectrum in Childhood Acute Lymphoblastic Leukemia.
      aug:
        au:
          Hicks, Chindo
          Sitthi-Amorn, Jitsuda
          Douglas, Jessica
          Ramani, Ritika
          Miele, Lucio
          Vijayakumar, Vani
          Karlson, Cynthia
          Chipeta, James
          Megason, Gail
        affil: Department of Genetics, Louisiana State University Health Sciences Center, New Orleans, LA, USA
      sug:
        subj:
          Leukemia, Lymphocytic, Acute
          Central Nervous System Diseases
          Gene Expression
          B Lymphocytes
          Central Nervous System
          Child
          Data Analysis
          Clinical Trials
          Study Design
          Probability
          Reliability
          Child: 6-12 years
      ab: Treatment of the central nervous system (CNS) is an essential therapeutic component in childhood acute lymphoblastic leukemia (ALL). The goal of this study was to identify molecular signatures distinguishing patients with CNS disease from those without the disease in pediatric patients with ALL. We analyzed gene expression data from 207 pediatric patients with ALL. Patients without CNS were classified as CNS1, while those with mild and advanced CNS disease were classified as CNS2 and CNS3, respectively. We compared gene expression levels among the three disease classes. We identified gene signatures distinguishing the three disease classes. Pathway analysis revealed molecular networks and biological pathways dysregulated in response to CNS disease involvement. The identified pathways included the ILK, WNT, B-cell receptor, AMPK, ERK5, and JAK signaling pathways. The results demonstrate that transcription profiling could be used to stratify patients to guide therapeutic decision-making in pediatric ALL.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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