Overexpression of Rsf-1 correlates with poor survival and promotes invasion in non-small cell lung cancer.

Rsf-1 (HBXAP) was recently reported to play roles in tumorigenesis and tumor progression. There have been many reports referred to Rsf-1 overexpression in various cancers and associated with the malignant behavior of cancer cells. However, the molecular mechanism of Rsf-1 in non-small cell lung canc...

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Publicado en:Virchows Archiv: European Journal of Pathology Vol. 470; no. 5; pp. 553 - 561
Autores principales: Zhang, Xiuwei, Fu, Lin, Xue, Dongwei, Zhang, Xiupeng, Hao, Fengxia, Xie, Lingling, He, Jiani, Gai, Junda, Liu, Yuhui, Xu, Hongtao, Li, Qingchang, Wang, Enhua
Formato: pictorial research tables/charts Journal Article
Publicado: Springer Nature May2017
Acceso en línea:Ver este registro en EBSCOhost
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        atl: Overexpression of Rsf-1 correlates with poor survival and promotes invasion in non-small cell lung cancer.
      aug:
        au:
          Zhang, Xiuwei
          Fu, Lin
          Xue, Dongwei
          Zhang, Xiupeng
          Hao, Fengxia
          Xie, Lingling
          He, Jiani
          Gai, Junda
          Liu, Yuhui
          Xu, Hongtao
          Li, Qingchang
          Wang, Enhua
        affil: Department of Pathology , The 1st Affiliated Hospital and College of Basic Medical Sciences of China Medical University , Shenyang 110001 People's Republic of China
      sug:
        subj:
          Lung Neoplasms Pathology
          Nuclear Proteins
          Proteins
          Carcinoma, Non-Small-Cell Lung Pathology
          Peptide Hydrolases Metabolism
          NF-kappa B Metabolism
          Lung Neoplasms Mortality
          Immunohistochemistry
          Neoplasm Invasiveness Pathology
          Genetic Techniques
          Blotting, Western
          Carcinoma, Non-Small-Cell Lung Mortality
          Prognosis
          Human
      ab: Rsf-1 (HBXAP) was recently reported to play roles in tumorigenesis and tumor progression. There have been many reports referred to Rsf-1 overexpression in various cancers and associated with the malignant behavior of cancer cells. However, the molecular mechanism of Rsf-1 in non-small cell lung cancer aggressiveness remains ambiguous. In the present study, we found that there was a significant association between Rsf-1 overexpression and poor overall survival (p = 0.028) in lung cancer. Furthermore, knockdown of Rsf-1 expression in H1299 and H460 cells with high endogenous Rsf-1 expression inhibited cell migration and invasion and downregulated MMP2 expression and nuclear levels of NF-κB. NF-κB inhibitor could also block the effect of Rsf-1 in regulation of MMP2 expression. Further experiments demonstrated that Rsf-1 depletion restrained NF-κB reporter luciferase activity and downregulated bcl-2 and p-IκB protein level. In conclusion, we demonstrated that Rsf-1 was overexpressed in lung cancer and associated with poor survival. Rsf-1 regulated cell invasion through MMP2 and NF-κB pathway.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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