Cortical thickness development of human primary visual cortex related to the age of blindness onset.

Blindness primarily induces structural alteration in the primary visual cortex (V1). Some studies have found that the early blind subjects had a thicker V1 compared to sighted controls, whereas late blind subjects showed no significant differences in the V1. This implies that the age of blindness on...

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Publicado en:Brain Imaging & Behavior Vol. 11; no. 4; pp. 1029 - 1037
Autores principales: Li, Qiaojun, Song, Ming, Xu, Jiayuan, Qin, Wen, Yu, Chunshui, Jiang, Tianzi
Formato: research Journal Article
Publicado: Springer Nature Aug2017
Acceso en línea:Ver este registro en EBSCOhost
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      pub: Springer Nature
      place: New York, New York
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        atl: Cortical thickness development of human primary visual cortex related to the age of blindness onset.
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        au:
          Li, Qiaojun
          Song, Ming
          Xu, Jiayuan
          Qin, Wen
          Yu, Chunshui
          Jiang, Tianzi
        affil: Department of Radiology , Tianjin Medical University General Hospital , Tianjin 300052 China
      sug:
        subj:
          Occipital Lobe
          Blindness
          Auditory Cortex Pathology
          Occipital Lobe Pathology
          Adult
          Dominance, Cerebral
          Parietal Lobe
          Blindness Physiopathology
          Blindness Epidemiology
          Frontal Lobe
          Auditory Cortex
          Image Processing, Computer Assisted
          Male
          Frontal Lobe Pathology
          Analysis of Variance
          Body Weights and Measures
          Parietal Lobe Pathology
          Blindness Pathology
          Age of Onset
          Human
          Magnetic Resonance Imaging
          Female
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
          Adult: 19-44 years
          Male
          Female
      ab: Blindness primarily induces structural alteration in the primary visual cortex (V1). Some studies have found that the early blind subjects had a thicker V1 compared to sighted controls, whereas late blind subjects showed no significant differences in the V1. This implies that the age of blindness onset may exert significant effects on the development of cortical thickness of the V1. However, no previous research used a trajectory of the age of blindness onset-related changes to investigate these effects. Here we explored this issue by mapping the cortical thickness trajectory of the V1 against the age of blindness onset using data from 99 blind individuals whose age of blindness onset ranged from birth to 34 years. We found that the cortical thickness of the V1 could be fitted well with a quadratic curve in both the left (F = 11.59, P = 3 × 10-5) and right hemispheres (F = 6.54, P = 2 × 10-3). Specifically, the cortical thickness of the V1 thinned rapidly during childhood and adolescence and did not change significantly thereafter. This trend was not observed in the primary auditory cortex (A1), primary motor cortex (M1), or primary somatosensory cortex (S1). These results provide evidence that an onset of blindness before adulthood significantly affects the cortical thickness of the V1 and suggest a critical period for cortical development of the human V1.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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