Gender Differences in the Progression of Experimental Chronic Kidney Disease Induced by Chronic Nitric Oxide Inhibition.

Chronic kidney disease (CKD) is considered a public health problem, assuming epidemic proportions worldwide. In this context, the preponderance of CKD prevalence in male over age-matched female patients is of note. In the present study, we investigated the impact of the gender on the development of...

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Publicado en:BioMed Research International Vol. 2017; pp. 1 - 13
Autores principales: Fanelli, Camilla, Dellê, Humberto, Cavaglieri, Rita Cassia, Dominguez, Wagner Vasques, Noronha, Irene L.
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 10/18/2017
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 10/18/2017
      vid: 2017
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2017/2159739
        125803065
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        atl: Gender Differences in the Progression of Experimental Chronic Kidney Disease Induced by Chronic Nitric Oxide Inhibition.
      aug:
        au:
          Fanelli, Camilla
          Dellê, Humberto
          Cavaglieri, Rita Cassia
          Dominguez, Wagner Vasques
          Noronha, Irene L.
        affil: Laboratory of Cellular, Genetic, and Molecular Nephrology, Renal Division, University of São Paulo, São Paulo, SP, Brazil
      sug:
        subj:
          Sex Factors
          Kidney Diseases
          Nitric Oxide
          Models, Biological
          Rats
          Vasoconstrictor Agents
          Albuminuria
          Renin-Angiotensin System
          Sex Hormones
      ab: Chronic kidney disease (CKD) is considered a public health problem, assuming epidemic proportions worldwide. In this context, the preponderance of CKD prevalence in male over age-matched female patients is of note. In the present study, we investigated the impact of the gender on the development of experimental CKD induced by chronic nitric oxide (NO) inhibition in Wistar male and female rats through the administration of L-NAME. CKD model induced by L-NAME is characterized by systemic vasoconstriction, resulting in severe hypertension, albuminuria, renal ischemia, glomerulosclerosis, interstitial expansion, and macrophage infiltration. After 30 days of CKD induction, male NAME rats exhibited remarkable albuminuria, augmented cortical histological damage, interstitial inflammation, and fibrosis. Age-matched female NAME rats showed significantly lower albuminuria, diminished glomerular ischemia, and glomerulosclerosis, as well as a significant reduction in the expression of α-smooth muscle actin renal interstitial Ang II+ cells. Thus, the present study demonstrated that female rats submitted to the NAME model developed less severe CKD than males. Female renoprotection could be promoted by both the estrogen anti-inflammatory activity and/or by the lack of testosterone, related to renin-angiotensin-aldosterone system hyperactivation and fibrogenesis. However, the influence of sex hormones on the progression of CKD needs to be further investigated.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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