Eplerenone might affect atrial fibrosis in patients with hypertension.

Background: Eplerenone is reported to reduce the development of atrial fibrillation (AF). The aim of this study was to clarify the mechanism of eplerenone for AF prevention from the viewpoint of P wavemorphology, which is reported to correlate with atrial fibrosis. Methods: Thirty-five patients with...

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Publicado en:Pacing & Clinical Electrophysiology Vol. 40; no. 10; pp. 1096 - 1103
Autores principales: Masato Kawasaki, Takahisa Yamada, Yuji Okuyama, Takashi Morita, Yoshio Furukawa, Shunsuke Tamaki, Yusuke Iwasaki, Atsushi Kikuchi, Yasushi Sakata, Masatake Fukunami
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell Oct2017
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Oct2017
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Eplerenone might affect atrial fibrosis in patients with hypertension.
      aug:
        au:
          Masato Kawasaki
          Takahisa Yamada
          Yuji Okuyama
          Takashi Morita
          Yoshio Furukawa
          Shunsuke Tamaki
          Yusuke Iwasaki
          Atsushi Kikuchi
          Yasushi Sakata
          Masatake Fukunami
        affil: Division of Cardiology, Osaka General Medical Center, Osaka, Japan.
      sug:
        subj:
          Eplerenone
          Hypertension Diagnosis
          Fibrosis Etiology
          Heart Atrium Pathology
          Atrial Fibrillation Drug Therapy
          Human
          Eplerenone Adverse Effects
      ab: Background: Eplerenone is reported to reduce the development of atrial fibrillation (AF). The aim of this study was to clarify the mechanism of eplerenone for AF prevention from the viewpoint of P wavemorphology, which is reported to correlate with atrial fibrosis. Methods: Thirty-five patients with hypertension, who were randomized to receive eplerenone (n = 16) or amlodipine (n = 19) for 1 year, were evaluated. P wave signal-averaged electrocardiography was recorded at baseline and 1 year after entry, and P wave duration (Ad) and P wave dispersion (P-disp) were obtained. Serum levels of intact procollagen type I N-terminal propeptide (PINP) and N-terminal procollagen-III peptide (PIIIP) were alsomeasured. Results: There were no significant differences in baseline clinical characteristics including Ad, P-disp, and the decrease in blood pressure at 1-year follow-up between the two groups. Ad and P-disp (mean ± standard deviation) significantly increased in patients on amlodipine after 1 year (140 ± 21 ms to 139 ± 19 ms vs 132 ± 10 ms to 136 ± 12 ms, P < 0.01 and 14 ± 7 ms to 9 ± 4 ms vs 12 ± 5 to 16 ± 8, P < 0.01, respectively). PINP was significantly more decreased in patients with eplerenone than amlodipine (56.6 ± 30.4 μg/mL to 46.6 ± 19.4 μg/mL vs 41.5 ± 16.2 μg/L to 48.7 ± 21.3 μg/L, P < 0.01). Percent changes of Ad, P-disp, PINP, and PIIIP were significantly smaller in patients with eplerenone than amlodipine (0.0 ± 4.7% vs 3.2 ± 4.4%, P < 0.05, - 28.6 ± 31.0% vs 46.3 ± 73.0%, P < 0.01, - 5.6 ± 38.1% vs 22.7 ± 42.7%, P < 0.05, and - 9.2 ± 25.1% vs 7.4 ± 19.0%, P < 0.05, respectively). Conclusions: Eplerenone reduced the increase of Ad and P-disp with a decrease of PINP and PIIIP, which might translate into reduction of atrial fibrosis. This study showed that eplerenone may be useful as upstream therapy for AF in patients with hypertension.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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