CXCR4 antagonist delivery on decellularized skin scaffold facilitates impaired wound healing in diabetic mice by increasing expression of SDF-1 and enhancing migration of CXCR4-positive cells.

C-X-C chemokine receptor type 4 (CXCR4) is an alpha-chemokine receptor specific for stromal cell-derived factor 1 (SDF-1 also called CXCL12). The antagonist of CXCR4 can mobilize CD34+ cells and hematopoietic stem cells from bone marrow within several hours, and it has an efficacy on diabetes ulcer...

Descripción completa

Detalles Bibliográficos
Publicado en:Wound Repair & Regeneration Vol. 25; no. 4; pp. 652 - 665
Autores principales: Liu, Hao, Liu, Hanping, Deng, Xiaoyuan, Chen, Maosheng, Han, Xue, Yan, Wenxia, Wang, Ning
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell Jul/Aug2017
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=126261534&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 126261534
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        10671927
        DPV
      jtl: Wound Repair & Regeneration
      issn: 10671927
      maglogo: Y
    pubinfo:
      dt: Jul/Aug2017
      vid: 25
      iid: 4
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
    artinfo:
      ui:
        126261534
        126261534
        126261534
        10.1111/wrr.12552
        126261534
      ppf: 652
      ppct: 13
      formats:
        fmt:
          – @attributes:
              type: T
          – @attributes:
              type: P
      tig:
        atl: CXCR4 antagonist delivery on decellularized skin scaffold facilitates impaired wound healing in diabetic mice by increasing expression of SDF-1 and enhancing migration of CXCR4-positive cells.
      aug:
        au:
          Liu, Hao
          Liu, Hanping
          Deng, Xiaoyuan
          Chen, Maosheng
          Han, Xue
          Yan, Wenxia
          Wang, Ning
        affil: College of Biophotonics, South China Normal University, Guangzhou China
      sug:
        subj:
          Wound Healing
          Diabetes Mellitus Diagnosis
          Gene Expression
          Animal Studies
          Mice
          Data Analysis Software
          Analysis of Variance
          Male
          Cell Migration Assays
          Cell Migration Inhibition
          Receptors, Cell Surface
          Chemokines
          Cell Physiology
          Matrix Metalloproteinases
          Skin Anatomy and Histology
          Protocols
          Male
      ab: C-X-C chemokine receptor type 4 (CXCR4) is an alpha-chemokine receptor specific for stromal cell-derived factor 1 (SDF-1 also called CXCL12). The antagonist of CXCR4 can mobilize CD34+ cells and hematopoietic stem cells from bone marrow within several hours, and it has an efficacy on diabetes ulcer through acting on the SDF-1/CXCR4 axis. In this study, we investigated for the first time whether the antagonist of CXCR4 (Plerixafor/AMD3100) delivered on acellular dermal matrix (ADM) may accelerate diabetes-impaired wound healing. ADM scaffolds were fabricated from nondiabetic mouse skin through decellularization processing and incorporated with AMD3100 to construct ADM-AMD3100 scaffold. Full-thickness cutaneous wound in streptozotocin (STZ)-induced diabetic mice were treated with ADM, AMD3100, or ADM-AMD3100. 21 days after treatment, wound closure in ADM-AMD3100-treated mice was more complete than ADM group and AMD3100 group, and it was accompanied by thicker collagen formation. Correspondingly, diabetic mice treated with ADM-AMD3100 demonstrated prominent neovascularization (higher capillary density and vascular smooth muscle actin), which were accompanied by up-regulated mRNA levels of SDF-1 and enhanced migration of CXCR4 in the granulation tissue. Our results demonstrate that ADM scaffold provide perfect niche for loading AMD3100 and ADM-AMD3100 is a promising method for diabetic wound healing mainly by increasing expression of SDF-1 and enhancing migration of CXCR4-positive cells.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N