Investigation of Programmed Cell Death-1 (PD-1) Gene Variations at Positions PD1.3 and PD1.5 in Iranian Patients with Non-small Cell Lung Cancer.

Background: Tumor cells express PD-1 ligands to bind PD-1 on immune cells and escape immune responses. In the present study, we aimed to investigate whether single nucleotide polymorphisms at positions PD1.3 (+7146, rs11568821) G/A, and PD1.5 (+7785 C/T, rs2227981) may be considered risk factors for...

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Publicado en:Middle East Journal of Cancer Vol. 9; no. 1; pp. 13 - 18
Autores principales: Pirdelkhosh, Zahra, Kazemi, Tohid, Haghshenas, Mohammad Reza, Ghayumi, Mohammad Ali, Erfani, Nasrollah
Formato: research tables/charts Journal Article
Publicado: Middle East Journal of Cancer Jan2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jan2018
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      pub: Middle East Journal of Cancer
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        atl: Investigation of Programmed Cell Death-1 (PD-1) Gene Variations at Positions PD1.3 and PD1.5 in Iranian Patients with Non-small Cell Lung Cancer.
      aug:
        au:
          Pirdelkhosh, Zahra
          Kazemi, Tohid
          Haghshenas, Mohammad Reza
          Ghayumi, Mohammad Ali
          Erfani, Nasrollah
        affil: Immunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
      sug:
        subj:
          Carcinoma, Non-Small-Cell Lung Familial and Genetic
          Polymorphism, Genetic
          Iran
          Human
          Disease Susceptibility Risk Factors
          Carcinoma, Non-Small-Cell Lung Pathology
          Age Factors
          Sex Factors
          Polymerase Chain Reaction Methods
          Genotype
          DNA
          Alleles
          Haplotypes
          Receptors, Cell Surface
      ab: Background: Tumor cells express PD-1 ligands to bind PD-1 on immune cells and escape immune responses. In the present study, we aimed to investigate whether single nucleotide polymorphisms at positions PD1.3 (+7146, rs11568821) G/A, and PD1.5 (+7785 C/T, rs2227981) may be considered risk factors for susceptibility to nonsmall cell lung cancer in the Iranian population. Methods: This study enrolled 206 histopathologically confirmed lung cancer patients and 173 age/sex matched healthy controls. We performed PCR-RFLP to determine the genotypes of the extracted genomic DNA. Results: The frequencies of PD1.3 GG, GA and AA genotypes were 171 (83%), 31 (15%) and 4 (1.9%) out of 206 patients, and 144 (83.2%), 26 (15%), and 3 (1.7%) out of 173 controls, respectively. The frequencies of PD1.5 CC, CT and TT genotypes were 78 (37.9%), 100 (48.5%), and 28 (13.6%) in patients, and 60 (34.7%), 89 (51.4%), and 24 (13.9%) in controls. There were no significant differences in genotype analysis between patients and controls at positions PD1.3 (P=0.98) or PD1.5 (P=0.80). No significant differences existed in the frequencies of alleles and haplotypes between the two groups (P>0.05). Conclusion: Our data have indicated no association between PD1.3 (+7146) G/A and PD1.5 (+7785) C/T with susceptibility to non-small cell lung cancer. Investigation of other PD1 genetic variations and emerged haplotypes are required to completely define the role of PD1 genetic variations in susceptibility to lung cancer.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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