TGF-α Overexpression in Breast Cancer Bone Metastasis and Primary Lesions and TGF-α Enhancement of Expression of Procancer Metastasis Cytokines in Bone Marrow Mesenchymal Stem Cells.

Bone metastasis (BM) is the advanced complication of breast cancer, while bone marrow-derived mesenchymal stem cells (BMSCs) in the microenvironment unclearly contribute to cancer metastasis. This study investigated potential roles of transforming growth factor- (TGF-) <italic>α</italic> in the inte...

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Publicado en:BioMed Research International Vol. 2018; pp. 1 - 11
Autores principales: Sun, Jingbo, Cui, Haiyan, Gao, Yanxin, Pan, Yangjian, Zhou, Kun, Huang, Jingzhan, Lan, Jin, Wei, Qingzhu, Liu, Xiaolong, Liu, Lixin, Xian, Cory J.
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2/8/2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2/8/2018
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2018/6565393
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        atl: TGF-α Overexpression in Breast Cancer Bone Metastasis and Primary Lesions and TGF-α Enhancement of Expression of Procancer Metastasis Cytokines in Bone Marrow Mesenchymal Stem Cells.
      aug:
        au:
          Sun, Jingbo
          Cui, Haiyan
          Gao, Yanxin
          Pan, Yangjian
          Zhou, Kun
          Huang, Jingzhan
          Lan, Jin
          Wei, Qingzhu
          Liu, Xiaolong
          Liu, Lixin
          Xian, Cory J.
        affil: Department of General Surgery, The Third Affiliated Hospital of Southern Medical University, Guangzhou, Guangdong 510630, China
      sug:
        subj:
          Cytokines
          Gene Expression
          Breast Neoplasms
          Bone Metastases
          Bone Marrow
          Stem Cells
          Neoplasm Metastasis
          Neoplasms, Fibrous Tissue
          Retrospective Design
          Macrophages
          Neoplastic Processes
          Human
          Interleukins
          Vascular Endothelial Growth Factors
          Growth Substances
      ab: Bone metastasis (BM) is the advanced complication of breast cancer, while bone marrow-derived mesenchymal stem cells (BMSCs) in the microenvironment unclearly contribute to cancer metastasis. This study investigated potential roles of transforming growth factor- (TGF-) <italic>α</italic> in the interaction between breast cancer and BMSCs in BM. Clinical cases of breast cancer with bone metastasis (BMBC), breast cancer without bone metastasis (Non-BM-BC), and benign fibroadenoma (Benign) were enlisted in a retrospective study. TGF-<italic>α</italic> was found obviously overexpressed in BM lesion of BMBC compared to primary lesion of both BMBC and Non-BM-BC (P<0.01), and TGF-<italic>α</italic> was higher in primary lesion of both BMBC and Non-BM-BC (P<0.01) than Benign group. Interestingly, TGF-<italic>α</italic> in nontumor tissues of both BMBC and Non-BM-BC was at a higher level than Benign group (P<0.01), and numbers of macrophages in nontumor tissues of both BMBC and Non-BM-BC (P<0.01) were higher than Benign group. Furthermore, in cultured human BMSCs, TGF-<italic>α</italic> stimulated production of procancer cytokines including IL-6, VEGF, FGF10, FGF17, and TGF-<italic>β</italic>1 in a dose-dependent manner. Thus, TGF-<italic>α</italic> in BC could potentially be an important signal of carcinogenesis and metastasis. Macrophages in the nontumor tissue of BC may not be protective but could promote cancer metastasis.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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