Dihydromyricetin Attenuates TNF-α-Induced Endothelial Dysfunction through miR-21-Mediated DDAH1/ADMA/NO Signal Pathway.

Accumulating studies demonstrate that dihydromyricetin (DMY), a compound extracted from Chinese traditional herb,<italic> Ampelopsis grossedentata</italic>, attenuates atherosclerotic process by improvement of endothelial dysfunction. However, the underlying mechanism remains poorly understood. Thus...

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Publicado en:BioMed Research International Vol. 2018; pp. 1 - 13
Autores principales: Yang, Dafeng, Tan, Shenglan, Yang, Zhousheng, Jiang, Pei, Qin, Caie, Yuan, Qiong, Dang, Ruili, Yao, Xiaoxia, Qu, Jian, Lu, Qiong, Xu, Ping, Zhang, Bikui, Xiang, Daxiong, Chen, Lei
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2/28/2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2/28/2018
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2018/1047810
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        atl: Dihydromyricetin Attenuates TNF-α-Induced Endothelial Dysfunction through miR-21-Mediated DDAH1/ADMA/NO Signal Pathway.
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        au:
          Yang, Dafeng
          Tan, Shenglan
          Yang, Zhousheng
          Jiang, Pei
          Qin, Caie
          Yuan, Qiong
          Dang, Ruili
          Yao, Xiaoxia
          Qu, Jian
          Lu, Qiong
          Xu, Ping
          Zhang, Bikui
          Xiang, Daxiong
          Chen, Lei
        affil: Department of Pharmacy, Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China
      sug:
        subj:
          Flavonols Pharmacodynamics
          Drugs, Chinese Herbal Pharmacodynamics
          Plants, Medicinal
          Tumor Necrosis Factor
          Endothelium Pathology
          MicroRNA Drug Effects
          Hydrolases Drug Effects
          Arginine Drug Effects
          Nitric Oxide Drug Effects
          Signal Transduction Drug Effects
          Endothelium Drug Effects
          Epithelial Cells
          Cell Movement
          Nitric Oxide Synthases Drug Effects
          Phosphorylation
          Gene Expression
          Enzyme Inhibitors
          Human
          Vascular Diseases Drug Therapy
          Flavonols Therapeutic Use
          Drugs, Chinese Herbal Therapeutic Use
      ab: Accumulating studies demonstrate that dihydromyricetin (DMY), a compound extracted from Chinese traditional herb,<italic> Ampelopsis grossedentata</italic>, attenuates atherosclerotic process by improvement of endothelial dysfunction. However, the underlying mechanism remains poorly understood. Thus, the aim of this study is to investigate the potential mechanism behind the attenuating effects of DMY on tumor necrosis factor alpha- (TNF-<italic>α</italic>-) induced endothelial dysfunction. In response to TNF-<italic>α</italic>, microRNA-21 (miR-21) expression was significantly increased in human umbilical vein endothelial cells (HUVECs), in line with impaired endothelial dysfunction as evidenced by decreased tube formation and migration, endothelial nitric oxide synthase (eNOS) (ser1177) phosphorylation, dimethylarginine dimethylaminohydrolases 1 (DDAH1) expression and metabolic activity, and nitric oxide (NO) concentration as well as increased asymmetric dimethylarginine (ADMA) levels. In contrast, DMY or blockade of miR-21 expression ameliorated endothelial dysfunction in HUVECs treated with TNF-<italic>α</italic> through downregulation of miR-21 expression, whereas these effects were abolished by overexpression of miR-21. In addition, using a nonspecific NOS inhibitor, L-NAME, also abrogated the attenuating effects of DMY on endothelial dysfunction. Taken together, these data demonstrated that miR-21-mediated DDAH1/ADMA/NO signal pathway plays an important role in TNF-<italic>α</italic>-induced endothelial dysfunction, and DMY attenuated endothelial dysfunction induced by TNF-<italic>α</italic> in a miR-21-dependent manner.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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