Positron emission tomography assessment of cerebral glucose metabolic rates in autism spectrum disorder and schizophrenia.

Several models have been proposed to account for observed overlaps in clinical features and genetic predisposition between schizophrenia and autism spectrum disorder. This study assessed similarities and differences in topological patterns and vectors of glucose metabolism in both disorders in refer...

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Publicado en:Brain Imaging & Behavior Vol. 12; no. 2; pp. 532 - 547
Autores principales: Mitelman, Serge A., Bralet, Marie-Cecile, Mehmet Haznedar, M., Hollander, Eric, Shihabuddin, Lina, Hazlett, Erin A., Buchsbaum, Monte S.
Formato: Journal Article
Publicado: Springer Nature Apr2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2018
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      pub: Springer Nature
      place: New York, New York
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        atl: Positron emission tomography assessment of cerebral glucose metabolic rates in autism spectrum disorder and schizophrenia.
      aug:
        au:
          Mitelman, Serge A.
          Bralet, Marie-Cecile
          Mehmet Haznedar, M.
          Hollander, Eric
          Shihabuddin, Lina
          Hazlett, Erin A.
          Buchsbaum, Monte S.
        affil: Departments of Psychiatry and Neuroscience, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, 10029, New York, NY, USA
      sug:
        subj:
          Schizophrenia Metabolism
          Brain Metabolism
          Glucose Metabolism
          Tomography, Emission-Computed
          Adult
          Radiopharmaceuticals
          Female
          Magnetic Resonance Imaging
          Brain
          Brain Mapping
          Male
          Social Behavior
          Fludeoxyglucose F 18
          Psychology
          Schizophrenia
          Learning
          Scales
          Social Readjustment Rating Scale
          Adult: 19-44 years
          Female
          Male
      ab: Several models have been proposed to account for observed overlaps in clinical features and genetic predisposition between schizophrenia and autism spectrum disorder. This study assessed similarities and differences in topological patterns and vectors of glucose metabolism in both disorders in reference to these models. Co-registered 18fluorodeoxyglucose PET and MRI scans were obtained in 41 schizophrenia, 25 ASD, and 55 healthy control subjects. AFNI was used to map cortical and subcortical regions of interest. Metabolic rates were compared between three diagnostic groups using univariate and multivariate repeated-measures ANOVA. Compared to controls, metabolic rates in schizophrenia subjects were decreased in the frontal lobe, anterior cingulate, superior temporal gyrus, amygdala and medial thalamic nuclei; rates were increased in the occipital cortex, hippocampus, basal ganglia and lateral thalamic nuclei. In ASD subjects metabolic rates were decreased in the parietal lobe, frontal premotor and eye-fields areas, and amygdala; rates were increased in the posterior cingulate, occipital cortex, hippocampus and basal ganglia. In relation to controls, subjects with ASD and schizophrenia showed opposite changes in metabolic rates in the primary motor and somatosensory cortex, anterior cingulate and hypothalamus; similar changes were found in prefrontal and occipital cortices, inferior parietal lobule, amygdala, hippocampus, and basal ganglia. Schizophrenia and ASD appear to be associated with a similar pattern of metabolic abnormalities in the social brain. Divergent maladaptive trade-offs, as postulated by the diametrical hypothesis of their evolutionary relationship, may involve a more circumscribed set of anterior cingulate, motor and somatosensory regions and the specific cognitive functions they subserve.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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