Young Blood Plasma Administration to Fight Alzheimer's Disease?

Despite decades of intensive research, no drugs can cure or even stabilize Alzheimer's disease (AD). Current pharmacological treatments only partially mask the symptoms while the disease progresses within the brain. Finding a preventive measure or a cure for people with AD is indeed a worldwide urge...

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Publicado en:Rejuvenation Research Vol. 21; no. 2; pp. 178 - 182
Autor principal: Aicardi, Giorgio
Formato: Journal Article
Publicado: Mary Ann Liebert, Inc. Apr2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2018
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      pub: Mary Ann Liebert, Inc.
      place: New Rochelle, New York
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        10.1089/rej.2017.1940
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        atl: Young Blood Plasma Administration to Fight Alzheimer's Disease?
      aug:
        au: Aicardi, Giorgio
        affil: Department for Life Quality Studies, University of Bologna, Bologna, Italy.
      sug:
        subj:
          Alzheimer's Disease Metabolism
          Blood Component Transfusion Methods
          Alzheimer's Disease Therapy
          Memory Disorders Prevention and Control
          Models, Biological
          Signal Transduction
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          Mice
          Animals
          Female
          Age Factors
          Male
          Female
      ab: Despite decades of intensive research, no drugs can cure or even stabilize Alzheimer's disease (AD). Current pharmacological treatments only partially mask the symptoms while the disease progresses within the brain. Finding a preventive measure or a cure for people with AD is indeed a worldwide urgent priority. A recent interesting study by T. Wyss-Coray's research group provides the first evidence that exposure to young blood or plasma can reverse some AD-related molecular and behavioral alterations. Heterochronic parabiosis (shared blood circulation) of AD transgenic mice with young healthy mice did not reduce amyloidosis and microglial activation in AD mice, but reversed the loss of synaptophysin and calbindin (critical synaptic proteins, indicators of cognitive decline in AD) in the dentate gyrus, and the abnormal expression, in the hippocampus, of many genes involved in key neuronal signaling pathways. Moreover, repeated intravenous administration of plasma from young healthy mice to AD mice reversed the excessive phosphorylation of hippocampal extracellular signal-regulated kinase (ERK), and improved spatial working memory and associative memory. Although observations in mouse models of AD might not necessarily extrapolate to humans, this preclinical study provides the first demonstration that young plasma has potential therapeutic properties, by ameliorating aspects of the disease that are present in AD patients. Clinical trials are already under way. If young plasma transfusion will be effective in AD patients, it will be important to identify the key factors responsible for the positive effects, as they might lead to the development of molecule interventions with a better efficacy/risk profile.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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